Macrophage Pathobiology in Immune-Mediated Inflammatory Disease (IMIDs)
A special issue of Genes (ISSN 2073-4425). This special issue belongs to the section "Human Genomics and Genetic Diseases".
Deadline for manuscript submissions: closed (25 April 2022) | Viewed by 4476
Special Issue Editors
Interests: immune-mediated inflammatory diseases; resolution of inflammation; macrophages; fibroblasts; functional genomics; epigenetics
Special Issue Information
Dear Colleagues,
In this Special Issue, we are looking for articles that address the functions of blood-derived infiltrating and tissue-resident macrophages in health and autoimmune and inflammatory diseases—in particular, articles that cover the roles of macrophages in chronic inflammation and tissue damage, acute inflammation and other tissue insults, the resolution of inflammation and tissue repair and the switch between inflammation and resolution. These questions can be answered from the perspectives of macrophage ontogeny, tissue-specific phenotypes and functions, macrophage plasticity and memory during chronic inflammation and resolution (in the context of tolerance and trained immunity) and epigenetics. We invite articles from diverse fields with strong macrophage biology research, including, but not restricted to, the following:
- Diseases of the nervous system: multiple sclerosis, neuromyelitis optica, myasthenia gravis, et cetera.
- Diseases of the intestine: inflammatory bowel disease, celiac disease.
- Diseases of the skin: atopic dermatitis, psoriasis, aberrant wound healing.
- Diseases of the joint: rheumatoid arthritis, psoriatic arthritis, osteoarthritis.
- Systemic lupus erythematosus, systemic sclerosis, age-related chronic inflammatory diseases, et cetera.
- Vascular inflammatory diseases: atherosclerosis and vasculitides.
Dr. Mojca Frank-Bertoncelj
Dr. Swathi Lingam
Guest Editors
Manuscript Submission Information
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Keywords
- macrophage pathobiology
- immune-mediated inflammatory diseases
- acute and chronic inflammation
- tissue damage
- resolution of inflammation
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