Pulmonary Fibrosis: What We Have Learnt from Other Tissues

A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Cell Biology and Pathology".

Deadline for manuscript submissions: closed (31 October 2022) | Viewed by 335

Special Issue Editor


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Guest Editor
Institute for Respiratory Health, University of Western Australia, Perth, Australia
Interests: pulmonary fibrosis; mesothelial cell biology; malignant mesothelioma; post-operative adhesions; cell signalling; macrophage biology; animal models of disease
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Special Issue Information

Dear Colleagues,

There is a growing interest from the pharmaceutical industry to develop drugs to treat fibrotic diseases. Until recently, diseases such as idiopathic pulmonary fibrosis have had no effective treatments. However, the development of pirfenidone and nintedanib has renewed enthusiasm for developing better drugs that not only slow down the fibrotic response but reverse fibrosis. However, to develop more effective treatments, we first need to better understand disease pathogenesis. There is a growing literature on the pathogenesis of lung fibrosis using cells, tissues and fluids from patients and animal models. There is also a vast amount of data generated from studies examining disease mechanisms in other fibrotic conditions. The focus of this Special Issue will be to examine what we have learnt about the mechanisms driving fibrosis in other tissues and diseases to help us delineate the mechanisms driving fibrosis in the lung.

Dr. Steven E. Mutsaers
Guest Editor

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