Advanced Research on Gestational Diabetes: Focus on Fetal and Placental Endothelial Function
A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Endocrinology and Metabolism Research".
Deadline for manuscript submissions: closed (31 July 2023) | Viewed by 2146
Special Issue Editors
Interests: inborn and aquired metabolic disorders; endothelial function; inflammation; fetal and adult endothelial cells; role of fetal sex in fetal programming
Special Issue Information
Dear Colleagues,
Influences in utero modulate the susceptibility to the development of non-communicable chronic diseases later in life. Accordingly, gestational diabetes (GDM) is associated with an increased risk for the development of cardiovascular diseases in the later life of the offspring. As a maternal metabolic disorder, GDM establishes cardiovascular risk factors in the fetal circulation, including insulin resistance and a subtle pro-inflammatory environment which affects placental and fetal endothelial function. A dysfunctional placental endothelium with altered vascular architecture, flow rate or vascular resistance may consequently affect fetal heart development and manifest in cardiac changes. Moreover, the GDM environment may program the endothelial function and dysfunction in the long term through epigenetic changes. Thus, intrauterine exposure to cardiovascular risk factors may program and shape the future risk for endothelial dysfunction and cardiovascular disease.
This Special Issue will address the associations between maternal diabetes in pregnancy and placental and fetal endothelial dysfunction. Molecular mechanisms underlying the intrauterine programming of endothelial and cardiac dysfunction culminating in cardiovascular disease in later life are particularly of interest. Experimental work, epidemiological studies and review articles are welcome. Both human and animal studies are encouraged.
Dr. Silvija Cvitic
Dr. Ursula Hiden
Guest Editors
Manuscript Submission Information
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Keywords
- gestational diabetes
- endothelial function
- programming
- placenta
- cardiovascular risk factors
- epigenetics
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