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Cardiogenetics, Volume 12, Issue 4

December 2022 - 2 articles

Cover Story: Spontaneous coronary artery dissection (SCAD) is a cause of myocardial infarction, especially in younger women without cardiovascular risk factors. Patient management and diagnostics are still largely based on retrospective and observational studies. Most patients with SCAD report chest pain and have elevated biomarkers with ECG findings. SCAD can lead to cardiogenic shock, ventricular arrhythmias and cardiac arrest and is commonly associated with fibromuscular dysplasia (FMD). Genetic associations are still in their infancy with this disease process. The BGN mutation has been recognized to be correlated with aortic aneurysm and aortic dissection. It has not yet been found to be associated with SCAD/FMD. This paper highlights the potential link between the BGN gene and SCAD/FMD. Further research looking at this association is warranted. View this paper

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Articles (2)

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Editorial

1 Citations

3,305 Views

2 Pages

The Expanding Spectrum of FLNC Cardiomyopathy

Emanuele Monda,
Martina Caiazza and
Giuseppe Limongelli

Cardiogenetics2022, 12(4), 276-277;https://doi.org/10.3390/cardiogenetics12040027

22 November 2022

Mutations in gene encoding filamin C (FLNC) have been historically associated with hypertrophic cardiomyopathy (HCM) and myofibrillar myopathy [...]

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Case Report

4,123 Views

6 Pages

Could the BGN Gene Be Pathogenic with Spontaneous Coronary Artery Dissection (SCAD) and Fibromuscular Dysplasia (FMD)?

Lexie Kolton,
Charlie Robin,
Jianfeng Xu,
Jun Wei,
Rupa Patil and
Jason Robin

Cardiogenetics2022, 12(4), 270-275;https://doi.org/10.3390/cardiogenetics12040026

9 October 2022

BACKGROUND. Spontaneous coronary artery dissection (SCAD) is a cause of myocardial infarction, especially in younger women without cardiovascular risk factors. Patient management and diagnostics are still largely based on retrospective and observatio...

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Cardiogenetics - ISSN 2035-8148

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