Advances in Environmental Epidemiology, Health and Lifestyle

A special issue of Epidemiologia (ISSN 2673-3986). This special issue belongs to the section "Environmental Epidemiology".

Deadline for manuscript submissions: 30 April 2026 | Viewed by 601

Special Issue Editor


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Guest Editor
Herbert Wertheim School of Public Health and Human Longevity Science, University of California San Diego (UCSD), San Diego, CA 92093, USA
Interests: air pollution; respiratory health; human health; environmental health and epidemiology

Special Issue Information

Dear Colleagues,

This Special Issue of Epidemiologia, entitled “Advances in Environmental Epidemiology, Health and Lifestyle”, aims to highlight innovative research exploring how environmental exposures intersect with social, behavioral, and lifestyle factors to influence human health across the life course. We welcome original research articles and reviews that utilize population-based, cohort, or longitudinal study designs to examine the health impacts of environmental pollutants—such as heavy metals, endocrine-disrupting chemicals, pesticides, and persistent organic pollutants (POPs).

In particular, we encourage the submission of studies focused on vulnerable populations, including children, pregnant women, and socioeconomically disadvantaged groups. Health outcomes of interest include neurodevelopment, anthropometric indicators, liver function biomarkers, lipid profiles, and inflammation-related biomarkers. Interdisciplinary contributions incorporating lifestyle factors and contextual determinants—such as housing conditions, educational attainment, and neighborhood environments—are also very welcome.

This Special Issue also seeks to advance methodological approaches in environmental epidemiology, including biomarker-based exposure assessments and multivariate statistical modeling techniques for complex exposure mixtures. Our goal is to foster scientific dialog on preventive strategies, health equity, and policy actions that can mitigate the health risks of environmental exposures—particularly in low- and middle-income settings.

Dr. Rajendra Prasad Parajuli
Guest Editor

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Keywords

  • environmental epidemiology
  • neurodevelopment and child health
  • lifestyle and psychosocial factors
  • birth cohorts and longitudinal studies
  • environmental exposures (metals, POPs, pesticides)
  • health disparities and vulnerable populations
  • liver, lipid, and inflammation biomarkers
  • multivariate exposure modeling
  • environmental justice
  • public health interventions

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Published Papers (1 paper)

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Research

13 pages, 359 KB  
Article
Population Attributable Fraction of Tobacco Use and Type 2 Diabetes Mellitus: An Analysis of the ENSANUT 2021
by Julio Cesar Campuzano, Jorge Martin Rodríguez, Luz Myriam Reynales, Anaid Hernández and Diana Carolina Urrego
Epidemiologia 2025, 6(4), 84; https://doi.org/10.3390/epidemiologia6040084 - 2 Dec 2025
Viewed by 243
Abstract
Background: Robust evidence demonstrates that tobacco use acts as a causal and, therefore, modifiable risk factor for the development of type 2 diabetes mellitus (T2DM). However, its specific population-level impact in Mexico has not yet been quantified. Objective: This study aimed to estimate [...] Read more.
Background: Robust evidence demonstrates that tobacco use acts as a causal and, therefore, modifiable risk factor for the development of type 2 diabetes mellitus (T2DM). However, its specific population-level impact in Mexico has not yet been quantified. Objective: This study aimed to estimate the population attributable fraction (PAF) of T2DM associated with tobacco use among Mexican adults, utilizing data from the 2021 National Health and Nutrition Survey (ENSANUT). Methods: A nested case–control analysis was conducted within the complex sampling design of the ENSANUT. Adults aged 20 years or older were included. Cases were defined as individuals with a self-reported medical diagnosed T2DM diagnosis; controls were individuals without T2DM. Exposure status was categorized as current person who smokes, former person who smokes, and never person who smokes. A logistic regression model was employed, adjusting for key covariates including age, sex, socioeconomic status, and comorbidities. The PAF was subsequently calculated using the Miettinen formula. Results: The adjusted PAF for T2DM attributable to smoking was 10.1% (95% CI: 4.07–14.97). This finding suggests that approximately one in eight T2DM cases could be prevented through the elimination of tobacco use. The association was more pronounced among men and individuals with a history of heavy tobacco use. Conclusion: The estimated PAF for T2DM due to tobacco use underscores the significant contribution of policies established within the WHO Framework Convention on Tobacco Control to the prevention of chronic diseases. The implementation and strengthening of such policies, including increased tobacco taxes, comprehensive smoking bans in public places, on-package warnings, and advertising prohibitions, would prove highly beneficial. These findings show a strong population-level association between tobacco use and T2DM, but causality cannot be established. Future longitudinal studies in Mexico are needed to confirm these results. Full article
(This article belongs to the Special Issue Advances in Environmental Epidemiology, Health and Lifestyle)
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