Autoimmune Disease and Malignancy of the Thyroid Gland in the COVID-19 Pandemic Era
A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Immunology and Immunotherapy".
Deadline for manuscript submissions: closed (31 August 2023) | Viewed by 232
Special Issue Editor
Special Issue Information
Dear Colleagues,
The first pandemic of the new century, known as COVID-19, poses significant risks for individuals and communities all across the globe. The World Health Organization has not yet declared the pandemic to be over. The SARS-CoV-2 virus utilizes the ACE2 receptor in conjunction with the transmembrane protease serine 2 (TMPRSS2) as the primary molecular complex that allows it to infect host cells and enter cells. It is interesting to note that the levels of ACE2 and TMPRSS2 expression in the thyroid gland are much higher than in the lungs because of this. When dealing with COVID-19, it is essential to keep in mind that there is a possibility of the thyroid being damaged. Following on from the previous two points, COVID-19 may be responsible for inflammation of the thyroid gland by inducing aberrant immune responses and a cytokine storm. Furthermore, there is accumulating evidence that the incidence and severity of thyroid disorders induced by COVID-19, such as thyrotoxicosis, hypothyroidism, and the transformation of pre-existing benign thyroid nodules into thyroid cancer, rose dramatically throughout the course of the pandemic. In light of this, research into the mechanisms through which COVID-19 increases vulnerability to thyroid autoimmune diseases and modifies thyroid cancer outcomes should receive great importance.
Dr. Iyad Hassan
Guest Editor
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Keywords
- COVID-19
- pandemic
- thyroid cancer
- autoimmune thyroid disease
- thyroiditis
- Graves’ disease
- thyroiditis
- thyrotoxicosis
- hypothyroidism
- thyroidectomy
- coronavirus
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