The Molecular Basis of the Immune Response in Pulmonary Fibrosis
A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Immunology and Immunotherapy".
Deadline for manuscript submissions: 30 June 2025 | Viewed by 1111
Special Issue Editors
Interests: immunology; pulmonary fibrosis; aging; senescence
Interests: immunology; pulmonary fibrosis; aging; senescence
Special Issue Information
Dear Colleagues,
Different molecular mechanisms have been associated with the development and progression of pulmonary fibrosis. Environmental chronic exposures induce repetitive microinjuries that cause an aberrant activation of the epithelial cells. There is an abnormal repair, mediated by the proliferation of fibroblasts, its activation into myofibroblasts, and the production of large amounts of extracellular matrix components. In addition, as an age-associated disease, there is an accumulation of senescent cells with a very active cytokine and extracellular matrix production. In this complex scenario, the lung immune response is a double-edged sword. While excess inflammation can result in further tissue damage and promote the fibrotic process, defects of immune surveillance and immune resolution allow for the accumulation of fibrosis and senescent cells. This Special Issue of Biomedicines aims to focus on recent advances identifying molecular mechanisms affecting the immune response in the fibrotic lung.
Dr. Tamara Cruz
Dr. Fernanda Hernandez
Guest Editors
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Keywords
- immune response
- fibrosis
- aging
- senescence
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