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Anatomia, Volume 5, Issue 1 (March 2026) – 4 articles

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10 pages, 1314 KB  
Review
Lateral Patellar Compression Syndrome: Surgical Techniques and Treatment
by Mason Nolan, Ethan Marting, Sarah Willard, James Applegate, Morgan Turnow, Taylor Manes and Benjamin C. Taylor
Anatomia 2026, 5(1), 4; https://doi.org/10.3390/anatomia5010004 - 22 Jan 2026
Viewed by 119
Abstract
Anterolateral knee pain is a common complaint that can be debilitating for patients if not treated properly. Lateral Patellar Compression Syndrome (LPCS), characterized by the maltracking of the patella with flexion, placing undue stress on the lateral patellar facet, is a common mechanism [...] Read more.
Anterolateral knee pain is a common complaint that can be debilitating for patients if not treated properly. Lateral Patellar Compression Syndrome (LPCS), characterized by the maltracking of the patella with flexion, placing undue stress on the lateral patellar facet, is a common mechanism causing anterolateral knee pain. Symptoms tend to be exacerbated with deep/prolonged flexion as the lateral patellar facet is compressed on the lateral trochlear groove of the femur. While conservative treatment methods are often sufficient, persistent pain may indicate surgical intervention to correct mechanical malalignment. The surgical treatment of LPCS is not widely agreed upon, with numerous techniques being practiced and no single procedure being considered optimal. This narrative review synthesizes the available literature on surgical techniques for LPCS treatment. A comprehensive search strategy was not employed, limiting the systematic nature of our findings. Full article
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18 pages, 5667 KB  
Opinion
The Building of the Triangular Locus of the Atrioventricular Node from Todaro to Tandler
by Marcos C. De Almeida
Anatomia 2026, 5(1), 3; https://doi.org/10.3390/anatomia5010003 - 21 Jan 2026
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Abstract
What is known today as the triangle of Koch (the triangular locus of the atrioventricular node, TLAVN) is bordered by the fibrous attachment of the septal cusp of tricuspid valve, the opening of the coronary sinus and tendon of valve of inferior vena [...] Read more.
What is known today as the triangle of Koch (the triangular locus of the atrioventricular node, TLAVN) is bordered by the fibrous attachment of the septal cusp of tricuspid valve, the opening of the coronary sinus and tendon of valve of inferior vena cava (TIVCV). This is a concept developed cumulatively by several exceptional anatomists. The literature was reviewed with a focus on the discovery of the atrioventricular node by Sunao Tawara (January 1906), its previous announcement by Tawara’s mentor Ludwig Aschoff (1905), and the contributions of the authors who described the other components of the triangular locus. Francesco Todaro discovered the TIVCV (1865); Tawara described the atrioventricular node and its relationship with the fibrous attachment of the septal cusp of tricuspid valve and the opening of the coronary sinus. The first description of assembling all components was provided by Arthur Keith (March 1906). Keith was also the first to consider the triangular locus as a useful landmark for identifying the atrial structures of the conduction system discovered by Tawara and Wilhelm His Jr. (1893). Julius Tandler named the TLAVN as Koch’s triangle (1913). Keith’s contributions to this topic have been particularly overlooked. The “triangular locus of the atrioventricular node” or “triangle of the atrioventricular node” are more instructive and impartial names. Full article
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11 pages, 2453 KB  
Case Report
A Case of Double Superior Vena Cava with a Rare Accessory Hemiazygos Arch Crossing over the Descending Aorta in a Male Body Donor
by Sandeep Silawal, Mustafa Kandemir, Franz Stelzl, Valentina Oberguggenberger, Kristinko Martinovic, Maria Kokozidou, Niels Hammer and Gundula Schulze-Tanzil
Anatomia 2026, 5(1), 2; https://doi.org/10.3390/anatomia5010002 - 2 Jan 2026
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Abstract
While performing a routine anatomical dissection on a male donor, undergraduate medical students observed an uncommon vascular anomaly: a persistent left superior vena cava (LSVC). Prior to the anatomical dissection, computed tomography (CT) images were obtained in an embalmed condition. Relevant anatomical structures [...] Read more.
While performing a routine anatomical dissection on a male donor, undergraduate medical students observed an uncommon vascular anomaly: a persistent left superior vena cava (LSVC). Prior to the anatomical dissection, computed tomography (CT) images were obtained in an embalmed condition. Relevant anatomical structures were measured using the JiveX DICOM Viewer. The left brachiocephalic vein (LBV) was present as a communicating vessel with a markedly reduced diameter between the LSVC and the right superior vena cava (RSVC). The diameters of RSVC and LSVC averaged 19.4 mm and 15.2 mm, respectively. The LSVC drained into a dilated coronary sinus (CS), which measured 22.7 mm in diameter. In addition, the left accessory hemiazygos vein collected the 2nd to 5th left intercostal veins, forming a small-caliber venous arch (2.1 mm in diameter) at the T5 vertebral level, which crossed anterior to the thoracic aorta, before draining into the LSVC. In comparison, the azygos venous arch on the right side is connected to the RSVC at T4. Knowledge of such venous variations through preoperative imaging—such as CT, MRI, or echocardiography—can be essential for procedural planning and for minimizing inadvertent complications. This case also highlights a dual approach, combining anatomical dissection with detailed CT analysis of the same specimen, which can both enhance undergraduate anatomical education and contribute to high-quality morphological research. Full article
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19 pages, 1445 KB  
Review
Clinical Insights into Zenker’s Diverticulum: Anatomy, Pathophysiology, Diagnosis, and Evolving Treatments
by Diego Panci, Francesco Carini, Riccardo Chiodo, Sabrina David, Francesco Cappello and Giovanni Tomasello
Anatomia 2026, 5(1), 1; https://doi.org/10.3390/anatomia5010001 - 28 Dec 2025
Viewed by 546
Abstract
Background/Objectives: Zenker’s diverticulum (ZD) is a rare but clinically relevant condition. It is a false, pulsion-type diverticulum due to the protrusion of mucosal and submucosal layers through the Killian’s Triangle. Its pathogenesis is multifactorial and entails cricopharyngeus muscle dysfunction and age-related tissue [...] Read more.
Background/Objectives: Zenker’s diverticulum (ZD) is a rare but clinically relevant condition. It is a false, pulsion-type diverticulum due to the protrusion of mucosal and submucosal layers through the Killian’s Triangle. Its pathogenesis is multifactorial and entails cricopharyngeus muscle dysfunction and age-related tissue degeneration. This review addresses the current evidence regarding the anatomy, pathophysiology, clinical presentation, diagnostic approach, and therapeutic management of ZD. Methods: For this literature review, we searched the PubMed and Scopus databases using combinations of keywords relevant to Zenker’s diverticulum, including “Zenker’s diverticulum,” “esophageal diverticula,” “diagnosis,” “endoscopic treatment,” and “surgery”. We included articles published in recent decades, with a focus on most recent ones regarding clinical studies, systematic reviews, meta-analyses, and descriptions of new diagnostic and therapeutic techniques. Results: Characteristic symptoms comprise progressive dysphagia, regurgitation of undigested food, halitosis, and, in advanced cases, aspiration-related respiratory complications. Diagnosis of ZD is primarily based on barium swallow esophagography and endoscopic evaluation, complemented by other imaging techniques. Current therapeutic options include traditional open surgery and endoscopic procedures, including newer minimally invasive techniques. Conclusions: ZD is the most common type of esophageal diverticulum and can have a disabling impact on a patient’s quality of life. It is commonly underdiagnosed or misdiagnosed as another condition, and prevalence is expected to increase with the growing population ageing. Improved understanding of its pathophysiology is needed to refine diagnostic and therapeutic strategies and minimize recurrences and risks. Full article
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