Bamodu, O.A.; Chang, H.-L.; Ong, J.-R.; Lee, W.-H.; Yeh, C.-T.; Tsai, J.-T.
Elevated PDK1 Expression Drives PI3K/AKT/MTOR Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of Hepatocellular Carcinoma. Cells 2020, 9, 746.
https://doi.org/10.3390/cells9030746
AMA Style
Bamodu OA, Chang H-L, Ong J-R, Lee W-H, Yeh C-T, Tsai J-T.
Elevated PDK1 Expression Drives PI3K/AKT/MTOR Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of Hepatocellular Carcinoma. Cells. 2020; 9(3):746.
https://doi.org/10.3390/cells9030746
Chicago/Turabian Style
Bamodu, Oluwaseun Adebayo, Hang-Lung Chang, Jiann-Ruey Ong, Wei-Hwa Lee, Chi-Tai Yeh, and Jo-Ting Tsai.
2020. "Elevated PDK1 Expression Drives PI3K/AKT/MTOR Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of Hepatocellular Carcinoma" Cells 9, no. 3: 746.
https://doi.org/10.3390/cells9030746
APA Style
Bamodu, O. A., Chang, H.-L., Ong, J.-R., Lee, W.-H., Yeh, C.-T., & Tsai, J.-T.
(2020). Elevated PDK1 Expression Drives PI3K/AKT/MTOR Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of Hepatocellular Carcinoma. Cells, 9(3), 746.
https://doi.org/10.3390/cells9030746