Article Versions Notes

Diez ER, Sánchez JA, Prado NJ, Ponce Zumino AZ, García-Dorado D, Miatello RM, Rodríguez-Sinovas A. Ischemic Postconditioning Reduces Reperfusion Arrhythmias by Adenosine Receptors and Protein Kinase C Activation but Is Independent of K_ATP Channels or Connexin 43. International Journal of Molecular Sciences. 2019; 20(23):5927. https://doi.org/10.3390/ijms20235927

Diez, Emiliano Raúl, Jose Antonio Sánchez, Natalia Jorgelina Prado, Amira Zulma Ponce Zumino, David García-Dorado, Roberto Miguel Miatello, and Antonio Rodríguez-Sinovas. 2019. "Ischemic Postconditioning Reduces Reperfusion Arrhythmias by Adenosine Receptors and Protein Kinase C Activation but Is Independent of K_ATP Channels or Connexin 43" International Journal of Molecular Sciences 20, no. 23: 5927. https://doi.org/10.3390/ijms20235927

Diez, E. R., Sánchez, J. A., Prado, N. J., Ponce Zumino, A. Z., García-Dorado, D., Miatello, R. M., & Rodríguez-Sinovas, A. (2019). Ischemic Postconditioning Reduces Reperfusion Arrhythmias by Adenosine Receptors and Protein Kinase C Activation but Is Independent of K_ATP Channels or Connexin 43. International Journal of Molecular Sciences, 20(23), 5927. https://doi.org/10.3390/ijms20235927