Topic Editors
Skeletal Muscle Adaptations to Oxidative Stress
Topic Information
Dear Colleagues,
Intensive exercise may induce excessive stretching that cause ruptures of myofibril filaments, leading to a skeletal muscle loss of function through the failure of the excitation–contraction coupling system. These events generate an inflammatory response and a higher reactive oxygen species (ROS) production, thus compromising muscle function. ROS are continuously generated in the body and are usually promptly inactivated by cellular antioxidant defenses. In skeletal muscle, low concentrations of ROS modulate cell signaling processes and are required for normal force production. Conversely, higher ROS concentrations can lead to DNA, lipid, protein, and carbohydrate modifications, causing cellular function impairment and a reduced force production, thereby contributing to muscle fatigue. For these reasons, an assessment of the impact of exercise at both the molecular and the biochemical levels, as well as its effect on cellular signaling pathways, constitutes a crucial point of interest for the development of training protocols that are compatible with the health of individuals. This multidisciplinary topic, entitled “Skeletal Muscle Adaptations to Oxidative Stress”, invites researchers in this field to contribute by submitting original research or reviews, with an emphasis on describing new biomarkers or novel exercise-regulated signaling pathways, as well as new techniques and research approaches involved in the interplay between oxidative stress, physical activity, nutritional strategies, and skeletal muscle damage. The main focus of this topic is on human studies, but work with animal models will also be considered. Topics will include (but are not limited to) the following:
- Exercise protocols for health.
- Active life and sedentary lifestyle: the contribution of physical exercise.
- Aging and chronic diseases: exercise as medicine.
- Control of oxidative stress in exercise protocols.
- Physical activity and health of muscle tissue.
Dr. Guglielmo Duranti
Prof. Dr. Zsolt Radak
Topic Editors
Keywords
- oxidative stress
- exercise
- physical activity
- skeletal muscle
- muscle damage
- antioxidants
- reactive oxygen species
- redox balance
- aging
- nutrition
Participating Journals
| Journal Name | Impact Factor | CiteScore | Launched Year | First Decision (median) | APC | |
|---|---|---|---|---|---|---|
Antioxidants
|
8.2 | 14.7 | 2012 | 18.7 Days | CHF 2900 | Submit |
International Journal of Molecular Sciences
|
5.6 | 10.0 | 2000 | 17.5 Days | CHF 2900 | Submit |
Journal of Functional Morphology and Kinesiology
|
2.5 | 3.2 | 2016 | 20.1 Days | CHF 1800 | Submit |
Life
|
3.9 | 7.1 | 2011 | 15.3 Days | CHF 2600 | Submit |
Physiologia
|
2.1 | - | 2021 | 22.2 Days | CHF 1200 | Submit |
Preprints.org is a multidisciplinary platform offering a preprint service designed to facilitate the early sharing of your research. It supports and empowers your research journey from the very beginning.
MDPI Topics is collaborating with Preprints.org and has established a direct connection between MDPI journals and the platform. Authors are encouraged to take advantage of this opportunity by posting their preprints at Preprints.org prior to publication:
- Share your research immediately: disseminate your ideas prior to publication and establish priority for your work.
- Safeguard your intellectual contribution: Protect your ideas with a time-stamped preprint that serves as proof of your research timeline.
- Boost visibility and impact: Increase the reach and influence of your research by making it accessible to a global audience.
- Gain early feedback: Receive valuable input and insights from peers before submitting to a journal.
- Ensure broad indexing: Web of Science (Preprint Citation Index), Google Scholar, Crossref, SHARE, PrePubMed, Scilit and Europe PMC.
