The Molecular Footprint of Fetal Alcohol Spectrum Disorders and Prenatal Alcohol Exposure: Genetics, Epigenetics, Oxidative Stress and Neurodevelopement
A special issue of Genes (ISSN 2073-4425). This special issue belongs to the section "Epigenomics".
Deadline for manuscript submissions: 15 January 2026 | Viewed by 88
Special Issue Editor
Special Issue Information
Dear Colleagues,
Prenatal alcohol exposure (PAE) is a major environmental risk factor affecting fetal development, leading to a range of physical, cognitive, and behavioral impairments collectively known as Fetal Alcohol Spectrum Disorders (FASD). Recent advances in genetics, epigenomics, and molecular biology have shed light on the mechanisms by which alcohol disrupts neurodevelopment, yet many critical questions remain unanswered.
Individual genetic susceptibility, epigenetic modifications, and oxidative stress pathways all contribute to the diverse phenotypes observed in FASD. However, the complex interplay between these factors, and their role in long-term or even transgenerational outcomes, is still not fully understood. Identifying robust molecular biomarkers of exposure and outcome, understanding neuroinflammatory and neurotrophic signaling disruptions, and developing targeted interventions remain urgent goals.
In this Special Issue, we welcome original research, reviews, and methodological papers focusing on the molecular, genetic, epigenetic, oxidative, and neurodevelopmental consequences of PAE and their role in FASD. We aim to provide novel insights into mechanisms, diagnosis, and therapeutic strategies.
We look forward to your contributions.
Dr. Sergio Terracina
Guest Editor
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Keywords
- fetal alcohol spectrum disorders
- FASD
- molecular biology
- genetics
- epigenetics
- oxidative stress
- neuroinflammation
- neurodevelopment
- pathways
- diagnosis
- management
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