Gaikwad, A.V.; Eapen, M.S.; Dey, S.; Bhattarai, P.; Shahzad, A.M.; Chia, C.; Jaffar, J.; Westall, G.; Sutherland, D.; Singhera, G.K.;
et al. TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis (IPF): Role in Driving Endothelial-to-Mesenchymal Transition (EndMT). J. Clin. Med. 2024, 13, 1160.
https://doi.org/10.3390/jcm13041160
AMA Style
Gaikwad AV, Eapen MS, Dey S, Bhattarai P, Shahzad AM, Chia C, Jaffar J, Westall G, Sutherland D, Singhera GK,
et al. TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis (IPF): Role in Driving Endothelial-to-Mesenchymal Transition (EndMT). Journal of Clinical Medicine. 2024; 13(4):1160.
https://doi.org/10.3390/jcm13041160
Chicago/Turabian Style
Gaikwad, Archana Vijay, Mathew Suji Eapen, Surajit Dey, Prem Bhattarai, Affan Mahmood Shahzad, Collin Chia, Jade Jaffar, Glen Westall, Darren Sutherland, Gurpreet Kaur Singhera,
and et al. 2024. "TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis (IPF): Role in Driving Endothelial-to-Mesenchymal Transition (EndMT)" Journal of Clinical Medicine 13, no. 4: 1160.
https://doi.org/10.3390/jcm13041160
APA Style
Gaikwad, A. V., Eapen, M. S., Dey, S., Bhattarai, P., Shahzad, A. M., Chia, C., Jaffar, J., Westall, G., Sutherland, D., Singhera, G. K., Hackett, T.-L., Lu, W., & Sohal, S. S.
(2024). TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis (IPF): Role in Driving Endothelial-to-Mesenchymal Transition (EndMT). Journal of Clinical Medicine, 13(4), 1160.
https://doi.org/10.3390/jcm13041160
