Correction: Hui et al. Wrangling Actin Assemblies: Actin Ring Dynamics during Cell Wound Repair. Cells 2022, 11, 2777
Basic Sciences Division, Fred Hutchinson Cancer Center, Seattle, WA 98109, USA
*
Author to whom correspondence should be addressed.
Cells 2023, 12(11), 1532; https://doi.org/10.3390/cells12111532
Submission received: 29 March 2023
/
Accepted: 19 April 2023
/
Published: 2 June 2023
(This article belongs to the Special Issue Toward Understanding Wound Repair Mechanism)
In the original publication [1], there was a mistake in the legend for Figure 4. The correction of a typo was missed during the revision phase.
The correct legend of Figure 4 is as follows:
Figure 4. Branched and linear nucleation factors are crucial for cell wound repair. (A) Schematic diagram depicting crucial factors for stabilizing and nucleating linear actin filaments. Formins add actin monomers to the barbed end of the actin filament, whereas crosslinkers aid in bundling linear filaments. (B) Schematic diagram depicting branched filament assembly via the Arp2/3 complex and WAS family proteins. (C–F) XY super-resolution view of actin ring organization at 70% percent wound closure in control cell wounds showing a dense actin mesh circumscribing the wound (C), Diaphanous RNAi knockdowns (disrupting linear actin formation) exhibit a diffuse mesh of branched actin at the wound periphery (D), alpha-actinin RNAi knockdowns (an actin crosslinker needed for actin bundling) form a more sparse ring at the wound edge compared to that at control wounds (E), and Arp 2/3 RNAi knockdowns (disrupting branched actin formation) do not form an actin ring at the wound periphery, but rather have unusually long linear actin filaments within the wound (F). Scale bar: 5 µm. (G) Schematic diagram depicting different types of actin architectures. Reprinted from Ennomani et al., (2016), Curr. Biol. 26(5): 616–626. doi:10.1016/j.cub.2015.12.069 [60], with permission from Elsevier.
The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.
Reference
- Hui, J.; Stjepić, V.; Nakamura, M.; Parkhurst, S.M. Wrangling Actin Assemblies: Actin Ring Dynamics during Cell Wound Repair. Cells 2022, 11, 2777. [Google Scholar] [CrossRef] [PubMed]
Disclaimer/Publisher’s Note: The statements, opinions and data contained in all publications are solely those of the individual author(s) and contributor(s) and not of MDPI and/or the editor(s). MDPI and/or the editor(s) disclaim responsibility for any injury to people or property resulting from any ideas, methods, instructions or products referred to in the content. |
© 2023 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).
Share and Cite
MDPI and ACS Style
Hui, J.; Stjepić, V.; Nakamura, M.; Parkhurst, S.M. Correction: Hui et al. Wrangling Actin Assemblies: Actin Ring Dynamics during Cell Wound Repair. Cells 2022, 11, 2777. Cells 2023, 12, 1532. https://doi.org/10.3390/cells12111532
AMA Style
Hui J, Stjepić V, Nakamura M, Parkhurst SM. Correction: Hui et al. Wrangling Actin Assemblies: Actin Ring Dynamics during Cell Wound Repair. Cells 2022, 11, 2777. Cells. 2023; 12(11):1532. https://doi.org/10.3390/cells12111532
Chicago/Turabian StyleHui, Justin, Viktor Stjepić, Mitsutoshi Nakamura, and Susan M. Parkhurst. 2023. "Correction: Hui et al. Wrangling Actin Assemblies: Actin Ring Dynamics during Cell Wound Repair. Cells 2022, 11, 2777" Cells 12, no. 11: 1532. https://doi.org/10.3390/cells12111532
Note that from the first issue of 2016, this journal uses article numbers instead of page numbers. See further details here.
