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Correction to Int. J. Mol. Sci. 2019, 20(3), 502.
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Correction

Correction: Batsaikhan et al. Post-Injury Neuroprotective Effects of the Thalidomide Analog 3,6′-Dithiothalidomide on Traumatic Brain Injury. Int. J. Mol. Sci. 2019, 20, 502

by
Buyandelger Batsaikhan
1,
Jing-Ya Wang
1,
Michael T. Scerba
2,
David Tweedie
2,
Nigel H. Greig
2,
Jonathan P. Miller
3,
Barry J. Hoffer
3,
Chih-Tung Lin
1 and
Jia-Yi Wang
1,4,*
1
Graduate Institute of Medical Sciences, College of Medicine, Taipei Medical University, 250 Wu-Xing Street, Taipei 11031, Taiwan
2
Drug Design & Development Section, Translational Gerontology Branch, Intramural Research Program, National Institute on Aging, NIH, Baltimore, MD 21224, USA
3
Department of Neurological Surgery, Case Western Reserve University, Cleveland, OH 44106, USA
4
Department of Physiology, School of Medicine, College of Medicine, Taipei Medical University, Taipei 11031, Taiwan
*
Author to whom correspondence should be addressed.
Int. J. Mol. Sci. 2026, 27(1), 393; https://doi.org/10.3390/ijms27010393 (registering DOI)
Submission received: 25 November 2025 / Accepted: 27 November 2025 / Published: 30 December 2025
(This article belongs to the Section Molecular Pathology, Diagnostics, and Therapeutics)
Browse Figure
Versions Notes

Error in Figure

The authors wish to make the following corrections to this paper [1]:
The authors have realized an unfortunate error in the selected representative image for Sham > and TBI + Veh animals. The corrected Figure 5B appears below.
Corresponding to the corrected figure panel, the authors added statistical values (mean ± SEM) in the statement explaining Figure 5C. A correction has been made to the Section 2.5, the 9th Sentence: The number of Annexin V/NeuN positive cells was determined (yellow fluorescence) in the Sham group (13 ± 1), TBI + Veh group (184 ± 30), and TBI + 3,6′-DT group (117 ± 10) cells/mm2 (Figure 5C).
The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Batsaikhan, B.; Wang, J.-Y.; Scerba, M.T.; Tweedie, D.; Greig, N.H.; Miller, J.P.; Hoffer, B.J.; Lin, C.-T.; Wang, J.-Y. Post-Injury Neuroprotective Effects of the Thalidomide Analog 3,6′-Dithiothalidomide on Traumatic Brain Injury. Int. J. Mol. Sci. 2019, 20, 502. [Google Scholar] [CrossRef] [PubMed]
Ijms 27 00393 g005
Figure 5. Post-injury administration of 3,6′-DT at 5 h after TBI significantly decreased apoptotic neurons in the cortical contusion regions at 24 h. (A) The representative HE-stained coronal section showing the area as indicated by the black square box to compare the fluorescent signals across groups of rats. (B) The immunofluorescence of Annexin V and NeuN in cortical brain tissue. The Annexin V immunoreactivity is shown in green, and NeuN (a marker for neurons) is shown in red. The yellow color indicates colocalization. (C) The number of Annexin V/NeuN positive cells was elevated when evaluated at 24 h after TBI. Treatment with 3,6′-DT significantly decreased the number of apoptotic neurons compared with TBI + Veh animals. Data are presented as mean ± S.E.M. (n = 5 in each group). *** p < 0.001 compared with the Sham group. # p < 0.05 compared with the TBI + Veh group. Scale bar = 100 μm.
Figure 5. Post-injury administration of 3,6′-DT at 5 h after TBI significantly decreased apoptotic neurons in the cortical contusion regions at 24 h. (A) The representative HE-stained coronal section showing the area as indicated by the black square box to compare the fluorescent signals across groups of rats. (B) The immunofluorescence of Annexin V and NeuN in cortical brain tissue. The Annexin V immunoreactivity is shown in green, and NeuN (a marker for neurons) is shown in red. The yellow color indicates colocalization. (C) The number of Annexin V/NeuN positive cells was elevated when evaluated at 24 h after TBI. Treatment with 3,6′-DT significantly decreased the number of apoptotic neurons compared with TBI + Veh animals. Data are presented as mean ± S.E.M. (n = 5 in each group). *** p < 0.001 compared with the Sham group. # p < 0.05 compared with the TBI + Veh group. Scale bar = 100 μm.
Ijms 27 00393 g005
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MDPI and ACS Style

Batsaikhan, B.; Wang, J.-Y.; Scerba, M.T.; Tweedie, D.; Greig, N.H.; Miller, J.P.; Hoffer, B.J.; Lin, C.-T.; Wang, J.-Y. Correction: Batsaikhan et al. Post-Injury Neuroprotective Effects of the Thalidomide Analog 3,6′-Dithiothalidomide on Traumatic Brain Injury. Int. J. Mol. Sci. 2019, 20, 502. Int. J. Mol. Sci. 2026, 27, 393. https://doi.org/10.3390/ijms27010393

AMA Style

Batsaikhan B, Wang J-Y, Scerba MT, Tweedie D, Greig NH, Miller JP, Hoffer BJ, Lin C-T, Wang J-Y. Correction: Batsaikhan et al. Post-Injury Neuroprotective Effects of the Thalidomide Analog 3,6′-Dithiothalidomide on Traumatic Brain Injury. Int. J. Mol. Sci. 2019, 20, 502. International Journal of Molecular Sciences. 2026; 27(1):393. https://doi.org/10.3390/ijms27010393

Chicago/Turabian Style

Batsaikhan, Buyandelger, Jing-Ya Wang, Michael T. Scerba, David Tweedie, Nigel H. Greig, Jonathan P. Miller, Barry J. Hoffer, Chih-Tung Lin, and Jia-Yi Wang. 2026. "Correction: Batsaikhan et al. Post-Injury Neuroprotective Effects of the Thalidomide Analog 3,6′-Dithiothalidomide on Traumatic Brain Injury. Int. J. Mol. Sci. 2019, 20, 502" International Journal of Molecular Sciences 27, no. 1: 393. https://doi.org/10.3390/ijms27010393

APA Style

Batsaikhan, B., Wang, J.-Y., Scerba, M. T., Tweedie, D., Greig, N. H., Miller, J. P., Hoffer, B. J., Lin, C.-T., & Wang, J.-Y. (2026). Correction: Batsaikhan et al. Post-Injury Neuroprotective Effects of the Thalidomide Analog 3,6′-Dithiothalidomide on Traumatic Brain Injury. Int. J. Mol. Sci. 2019, 20, 502. International Journal of Molecular Sciences, 27(1), 393. https://doi.org/10.3390/ijms27010393

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