Mitochondria as Potential Target in Disease Therapy
A special issue of Future Pharmacology (ISSN 2673-9879).
Deadline for manuscript submissions: closed (10 December 2022) | Viewed by 4012
Special Issue Editors
Interests: mitochondria; reactive oxygen species; antioxidants
Special Issues, Collections and Topics in MDPI journals
Interests: cardiac reperfusion damage; mitochondrial bioenergetics; signal transduction; mitochondrial permeability transition pore
Interests: mitochondrial function; bioenergetics; redox signaling; chronic kidney disease; metabolic reprograming and extracelular vesicles
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
The current special issue is designated to publish original articles investigating mitochondria as a therapeutic target in diseases because relevance of the impairment in this organelle in metabolism, immunity, apoptosis, and signal transduction alterations.
Mitochondria are highly dynamic organelles that have a critical role in ROS production, cross talking between redox signaling and metabolic regulation, in cell death regulation and calcium homeostasis. Thus, mitochondria impairment plays a pivotal role in the genesis and development of acute and chronic injuries, triggering pathological pathways like inflammation, oxidative stress and fibrosis. Furthermore, recent use of mitochondrial target therapies has showed promising results in resolve or prevent different pathologies. Therefore, we invite investigators to submit an original research paper or review articles for publication in this Special issue. We are particularly interested in articles related to mitochondrial improvement in diseases such as metabolic diseases, cancer, kidney diseases, hepatic diseases, heart diseases, and more.
Prof. Dr. José Pedraza Chaverri
Prof. Dr. Cecilia Zazueta
Dr. Omar Emiliano Aparicio Trejo
Guest Editors
Manuscript Submission Information
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Keywords
- mitochondrial dysfunction
- mitochondrial therapeutic targets
- mitochondria and redox signaling
- metabolic regulation and mitochondria
- chronic and acute disease and mitochondria
- mitochondrial regulation in inflammation and fibrosis
- cell death and calcium regulation by mitochondria
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