Beta Cell Physiological Dynamics and Dysfunctional Transitions in Response to Islet Inflammation in Obesity and Diabetes
Abstract
1. Introduction
2. Systemic and Islet Inflammation in Obesity and Diabetes
3. Mediators of Islet Inflammation
4. Metabolic Interplay of Islet Inflammation, Glucolipotoxicity, and Beta Cell Dysfunction
5. Beta Cell Dysfunctional Transitions
5.1. Beta Cell Physiology (Optimal Beta Cell Function)
5.2. Beta Cell Hyperfunction
5.3. Beta Cell Hypofunction
5.4. Beta Cell Failure and Diabetes
6. Conclusions
Funding
Conflicts of Interest
References
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Cerf, M.E. Beta Cell Physiological Dynamics and Dysfunctional Transitions in Response to Islet Inflammation in Obesity and Diabetes. Metabolites 2020, 10, 452. https://doi.org/10.3390/metabo10110452
Cerf ME. Beta Cell Physiological Dynamics and Dysfunctional Transitions in Response to Islet Inflammation in Obesity and Diabetes. Metabolites. 2020; 10(11):452. https://doi.org/10.3390/metabo10110452
Chicago/Turabian StyleCerf, Marlon E. 2020. "Beta Cell Physiological Dynamics and Dysfunctional Transitions in Response to Islet Inflammation in Obesity and Diabetes" Metabolites 10, no. 11: 452. https://doi.org/10.3390/metabo10110452
APA StyleCerf, M. E. (2020). Beta Cell Physiological Dynamics and Dysfunctional Transitions in Response to Islet Inflammation in Obesity and Diabetes. Metabolites, 10(11), 452. https://doi.org/10.3390/metabo10110452