Do, T.T.; RodrÃguez-Beltran, J.; Cebrián-Sastre, E.; RodrÃguez-Rojas, A.; Castañeda-GarcÃa, A.; Blázquez, J.
Inactivation of a New Potassium Channel Increases Rifampicin Resistance and Induces Collateral Sensitivity to Hydrophilic Antibiotics in Mycobacterium smegmatis. Antibiotics 2022, 11, 509.
https://doi.org/10.3390/antibiotics11040509
AMA Style
Do TT, RodrÃguez-Beltran J, Cebrián-Sastre E, RodrÃguez-Rojas A, Castañeda-GarcÃa A, Blázquez J.
Inactivation of a New Potassium Channel Increases Rifampicin Resistance and Induces Collateral Sensitivity to Hydrophilic Antibiotics in Mycobacterium smegmatis. Antibiotics. 2022; 11(4):509.
https://doi.org/10.3390/antibiotics11040509
Chicago/Turabian Style
Do, Thi Thuy, Jerónimo RodrÃguez-Beltran, Esmeralda Cebrián-Sastre, Alexandro RodrÃguez-Rojas, Alfredo Castañeda-GarcÃa, and Jesús Blázquez.
2022. "Inactivation of a New Potassium Channel Increases Rifampicin Resistance and Induces Collateral Sensitivity to Hydrophilic Antibiotics in Mycobacterium smegmatis" Antibiotics 11, no. 4: 509.
https://doi.org/10.3390/antibiotics11040509
APA Style
Do, T. T., RodrÃguez-Beltran, J., Cebrián-Sastre, E., RodrÃguez-Rojas, A., Castañeda-GarcÃa, A., & Blázquez, J.
(2022). Inactivation of a New Potassium Channel Increases Rifampicin Resistance and Induces Collateral Sensitivity to Hydrophilic Antibiotics in Mycobacterium smegmatis. Antibiotics, 11(4), 509.
https://doi.org/10.3390/antibiotics11040509
