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Correction to Cells 2024, 13(3), 255.
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Correction

Correction: Li et al. Age-Associated Differences in Recovery from Exercise-Induced Muscle Damage. Cells 2024, 13, 255

by
Donna Ching Wah Li
1,2,
Stefan Rudloff
3,
Henning Tim Langer
4,
Kristina Norman
1,2,3,5,* and
Catrin Herpich
1,2,3
1
Department of Nutrition and Gerontology, German Institute of Human Nutrition Potsdam-Rehbrücke, 14558 Nuthetal, Germany
2
Institute of Nutritional Science, University of Potsdam, 14558 Nuthetal, Germany
3
Department of Geriatrics and Medical Gerontology, Charité–Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin and Humboldt-Universität zu Berlin, 13347 Berlin, Germany
4
Department of Medicine, Weill Cornell Medicine, New York, NY 10065, USA
5
German Center for Cardiovascular Research (DZHK), Partner Site Berlin, 10785 Berlin, Germany
*
Author to whom correspondence should be addressed.
Cells 2025, 14(19), 1564; https://doi.org/10.3390/cells14191564
Submission received: 16 August 2025 / Accepted: 3 September 2025 / Published: 9 October 2025
Browse Figure
Versions Notes

Error in Figure

In the original publication [1], there was a mistake in Figure 1 as published. The figure was changed during the revision process. An old figure was uploaded by mistake, which did not indicate the differences between older and younger adults. The corrected version of Figure 1 appears below. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Li, D.C.W.; Rudloff, S.; Langer, H.T.; Norman, K.; Herpich, C. Age-Associated Differences in Recovery from Exercise-Induced Muscle Damage. Cells 2024, 13, 255. [Google Scholar] [CrossRef] [PubMed]
Cells 14 01564 g001
Figure 1. Schematic comparison of the cellular response kinetics of post damaging exercise in young (line) and old humans (dotted line): extracellular matrix (ECM) changes (upper panel), indirect markers (middle panel), and inflammatory expression (lower panel). Upper panel: Tenascin-C (TN) and fibronectin (FN) are part of the transitional matrix and are thought to direct early satellite cell movement. Subsequently, collagens (COL) 1 and 3 and their matrix metalloproteinases (MMPs) increase for ECM remodeling. COL 4 is, an important component of the basement membrane, is thought to be remodeled during later stages. Middle panel: Decreases in maximal voluntary contraction (MVC) are prevalent immediately following exercise. Delayed onset muscle soreness (DOMS) peaks at around 24 h. Lower panel: Polymorphonuclear leukocytes (PMN) begin infiltrating the muscle immediately after the cessation of exercise. These differentiate into macrophages that ingest debris and apoptotic neutrophils. The production of local pro-inflammatory cytokines triggers the phenotypic switch from M1 macrophages to alternatively activated M2 macrophages. The early stage represents the inflammation phase, while the later stage indicates the beginning of the resolution phase. This figure is in part based on [42,51].
Figure 1. Schematic comparison of the cellular response kinetics of post damaging exercise in young (line) and old humans (dotted line): extracellular matrix (ECM) changes (upper panel), indirect markers (middle panel), and inflammatory expression (lower panel). Upper panel: Tenascin-C (TN) and fibronectin (FN) are part of the transitional matrix and are thought to direct early satellite cell movement. Subsequently, collagens (COL) 1 and 3 and their matrix metalloproteinases (MMPs) increase for ECM remodeling. COL 4 is, an important component of the basement membrane, is thought to be remodeled during later stages. Middle panel: Decreases in maximal voluntary contraction (MVC) are prevalent immediately following exercise. Delayed onset muscle soreness (DOMS) peaks at around 24 h. Lower panel: Polymorphonuclear leukocytes (PMN) begin infiltrating the muscle immediately after the cessation of exercise. These differentiate into macrophages that ingest debris and apoptotic neutrophils. The production of local pro-inflammatory cytokines triggers the phenotypic switch from M1 macrophages to alternatively activated M2 macrophages. The early stage represents the inflammation phase, while the later stage indicates the beginning of the resolution phase. This figure is in part based on [42,51].
Cells 14 01564 g001
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MDPI and ACS Style

Li, D.C.W.; Rudloff, S.; Langer, H.T.; Norman, K.; Herpich, C. Correction: Li et al. Age-Associated Differences in Recovery from Exercise-Induced Muscle Damage. Cells 2024, 13, 255. Cells 2025, 14, 1564. https://doi.org/10.3390/cells14191564

AMA Style

Li DCW, Rudloff S, Langer HT, Norman K, Herpich C. Correction: Li et al. Age-Associated Differences in Recovery from Exercise-Induced Muscle Damage. Cells 2024, 13, 255. Cells. 2025; 14(19):1564. https://doi.org/10.3390/cells14191564

Chicago/Turabian Style

Li, Donna Ching Wah, Stefan Rudloff, Henning Tim Langer, Kristina Norman, and Catrin Herpich. 2025. "Correction: Li et al. Age-Associated Differences in Recovery from Exercise-Induced Muscle Damage. Cells 2024, 13, 255" Cells 14, no. 19: 1564. https://doi.org/10.3390/cells14191564

APA Style

Li, D. C. W., Rudloff, S., Langer, H. T., Norman, K., & Herpich, C. (2025). Correction: Li et al. Age-Associated Differences in Recovery from Exercise-Induced Muscle Damage. Cells 2024, 13, 255. Cells, 14(19), 1564. https://doi.org/10.3390/cells14191564

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