Bernardo, B.C.; Yildiz, G.S.; Kiriazis, H.; Harmawan, C.A.; Tai, C.M.K.; Ritchie, R.H.; McMullen, J.R.
In Vivo Inhibition of miR-34a Modestly Limits Cardiac Enlargement and Fibrosis in a Mouse Model with Established Type 1 Diabetes-Induced Cardiomyopathy, but Does Not Improve Diastolic Function. Cells 2022, 11, 3117.
https://doi.org/10.3390/cells11193117
AMA Style
Bernardo BC, Yildiz GS, Kiriazis H, Harmawan CA, Tai CMK, Ritchie RH, McMullen JR.
In Vivo Inhibition of miR-34a Modestly Limits Cardiac Enlargement and Fibrosis in a Mouse Model with Established Type 1 Diabetes-Induced Cardiomyopathy, but Does Not Improve Diastolic Function. Cells. 2022; 11(19):3117.
https://doi.org/10.3390/cells11193117
Chicago/Turabian Style
Bernardo, Bianca C., Gunes S. Yildiz, Helen Kiriazis, Claudia A. Harmawan, Celeste M. K. Tai, Rebecca H. Ritchie, and Julie R. McMullen.
2022. "In Vivo Inhibition of miR-34a Modestly Limits Cardiac Enlargement and Fibrosis in a Mouse Model with Established Type 1 Diabetes-Induced Cardiomyopathy, but Does Not Improve Diastolic Function" Cells 11, no. 19: 3117.
https://doi.org/10.3390/cells11193117
APA Style
Bernardo, B. C., Yildiz, G. S., Kiriazis, H., Harmawan, C. A., Tai, C. M. K., Ritchie, R. H., & McMullen, J. R.
(2022). In Vivo Inhibition of miR-34a Modestly Limits Cardiac Enlargement and Fibrosis in a Mouse Model with Established Type 1 Diabetes-Induced Cardiomyopathy, but Does Not Improve Diastolic Function. Cells, 11(19), 3117.
https://doi.org/10.3390/cells11193117