Huang, C.-Y.; Deng, J.-S.; Huang, W.-C.; Jiang, W.-P.; Huang, G.-J.
Attenuation of Lipopolysaccharide-Induced Acute Lung Injury by Hispolon in Mice, Through Regulating the TLR4/PI3K/Akt/mTOR and Keap1/Nrf2/HO-1 Pathways, and Suppressing Oxidative Stress-Mediated ER Stress-Induced Apoptosis and Autophagy. Nutrients 2020, 12, 1742.
https://doi.org/10.3390/nu12061742
AMA Style
Huang C-Y, Deng J-S, Huang W-C, Jiang W-P, Huang G-J.
Attenuation of Lipopolysaccharide-Induced Acute Lung Injury by Hispolon in Mice, Through Regulating the TLR4/PI3K/Akt/mTOR and Keap1/Nrf2/HO-1 Pathways, and Suppressing Oxidative Stress-Mediated ER Stress-Induced Apoptosis and Autophagy. Nutrients. 2020; 12(6):1742.
https://doi.org/10.3390/nu12061742
Chicago/Turabian Style
Huang, Ching-Ying, Jeng-Shyan Deng, Wen-Chin Huang, Wen-Ping Jiang, and Guan-Jhong Huang.
2020. "Attenuation of Lipopolysaccharide-Induced Acute Lung Injury by Hispolon in Mice, Through Regulating the TLR4/PI3K/Akt/mTOR and Keap1/Nrf2/HO-1 Pathways, and Suppressing Oxidative Stress-Mediated ER Stress-Induced Apoptosis and Autophagy" Nutrients 12, no. 6: 1742.
https://doi.org/10.3390/nu12061742
APA Style
Huang, C.-Y., Deng, J.-S., Huang, W.-C., Jiang, W.-P., & Huang, G.-J.
(2020). Attenuation of Lipopolysaccharide-Induced Acute Lung Injury by Hispolon in Mice, Through Regulating the TLR4/PI3K/Akt/mTOR and Keap1/Nrf2/HO-1 Pathways, and Suppressing Oxidative Stress-Mediated ER Stress-Induced Apoptosis and Autophagy. Nutrients, 12(6), 1742.
https://doi.org/10.3390/nu12061742