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Correction to Int. J. Mol. Sci. 2021, 22(21), 11535.
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Correction

Correction: Shin et al. miR-29b Regulates TGF-β1-Induced Epithelial–Mesenchymal Transition by Inhibiting Heat Shock Protein 47 Expression in Airway Epithelial Cells. Int. J. Mol. Sci. 2021, 22, 11535

by
Jae-Min Shin
1,2,3,†,
Joo-Hoo Park
2,3,†,
Hyun-Woo Yang
2,3,
Jee Won Moon
1,
Heung-Man Lee
1,2 and
Il-Ho Park
1,2,3,*
1
Department of Otorhinolaryngology-Head and Neck Surgery, Korea University College of Medicine, Seoul 08308, Republic of Korea
2
Upper Airway Chronic Inflammatory Diseases Laboratory, Korea University College of Medicine, Seoul 08308, Republic of Korea
3
Medical Device Usability Test Center, Korea University Guro Hospital, Seoul 08308, Republic of Korea
*
Author to whom correspondence should be addressed.
†
These authors contributed equally to this work.
Int. J. Mol. Sci. 2025, 26(24), 11865; https://doi.org/10.3390/ijms262411865
Submission received: 20 November 2025 / Accepted: 27 November 2025 / Published: 9 December 2025
(This article belongs to the Section Macromolecules)
Browse Figures
Review Reports Versions Notes
In the original publication [1], there were errors in the +miR-29b mimic (α-SMA) in Figure 2f and +miR-29b (α-SMA) Control in Figure 3f; they were repeated. It appears that the α-SMA and E-cad data in Figure 2 were mistakenly left as temporary data and should have been updated. However, only the HSP data was corrected, while the α-SMA data was inadvertently not updated. And the labels in Figures 2c and 3c are incorrect. Figure 2c should be labeled as miR-29b mimic. Figure 3c should be labeled as miR-29b inhibitor. The corrected Figure 2c,f and Figure 3c appear below. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Shin, J.-M.; Park, J.-H.; Yang, H.-W.; Moon, J.W.; Lee, H.-M.; Park, I.-H. miR-29b Regulates TGF-β1-Induced Epithelial–Mesenchymal Transition by Inhibiting Heat Shock Protein 47 Expression in Airway Epithelial Cells. Int. J. Mol. Sci. 2021, 22, 11535. [Google Scholar] [CrossRef] [PubMed]
Ijms 26 11865 g002
Figure 2. Overexpression of miR-29b inhibited mRNA and protein expression levels of TGF-β1-induced EMT markers in A549 cells. A549 cells were stimulated with TGF-β1 (5 ng/mL) with miR control or miR-29b mimic. (a,b) mRNA expression levels of miR-29b and HSP47 were determined using qPCR. (c) HSP47 luciferase activity was measured by luciferase assay. (d) E-cadherin, α-SMA, vimentin, and fibronectin mRNA levels were analyzed through qPCR. (e) Protein expression levels of HSP47, E-cadherin, α-SMA, vimentin, and fibronectin were determined using Western blotting. (f) The cells were treated with TGF-β1 for 72 h after transfection of miR-29b mimic and then assessed for HSP47 (1st line, green), vimentin (1st line, red), α-SMA (2nd, green), and E-cadherin (2nd, red) expression/localization using immunofluorescence. Nuclei were stained with DAPI (blue). Scale bar = 20 μm. Values are expressed as mean ± SEM of three independent samples. * p < 0.05, vs. control + miR Control; † p < 0.05, vs. TGF-β1 + miR Control.
Figure 2. Overexpression of miR-29b inhibited mRNA and protein expression levels of TGF-β1-induced EMT markers in A549 cells. A549 cells were stimulated with TGF-β1 (5 ng/mL) with miR control or miR-29b mimic. (a,b) mRNA expression levels of miR-29b and HSP47 were determined using qPCR. (c) HSP47 luciferase activity was measured by luciferase assay. (d) E-cadherin, α-SMA, vimentin, and fibronectin mRNA levels were analyzed through qPCR. (e) Protein expression levels of HSP47, E-cadherin, α-SMA, vimentin, and fibronectin were determined using Western blotting. (f) The cells were treated with TGF-β1 for 72 h after transfection of miR-29b mimic and then assessed for HSP47 (1st line, green), vimentin (1st line, red), α-SMA (2nd, green), and E-cadherin (2nd, red) expression/localization using immunofluorescence. Nuclei were stained with DAPI (blue). Scale bar = 20 μm. Values are expressed as mean ± SEM of three independent samples. * p < 0.05, vs. control + miR Control; † p < 0.05, vs. TGF-β1 + miR Control.
Ijms 26 11865 g002
Ijms 26 11865 g003
Figure 3. Inhibition of miR-29b expression induced mRNA and protein expression levels of TGF-β1-induced EMT markers in A549 cells. A549 cells were stimulated with TGF-β1 (5 ng/mL) with miR control or a miR-29b inhibitor. (a,b) The mRNA expression levels of miR-29b and HSP47 were analyzed using qPCR. (c) HSP47 luciferase activity was measured by luciferase assay. (d) The mRNA levels of EMT-related markers were measured using qPCR. (e) Protein expression levels of HSP47, E-cadherin, α-SMA, vimentin and fibronectin were determined using Western blotting. (f) The cells were treated with TGF-β1 for 72 h after transfection with miR-29b inhibitor, and then assessed for HSP47 (1st line, green), vimentin (1st line, red), α-SMA (2nd, green), and E-cadherin (2nd, red) expression/localization using immunofluorescence. Nuclei were stained with DAPI (blue). Scale bar = 20 μm. Values are expressed as mean ± SEM of three independent samples. * p < 0.05 vs. control + miR Control; † p < 0.05, vs. TGF-β1 + miR Control.
Figure 3. Inhibition of miR-29b expression induced mRNA and protein expression levels of TGF-β1-induced EMT markers in A549 cells. A549 cells were stimulated with TGF-β1 (5 ng/mL) with miR control or a miR-29b inhibitor. (a,b) The mRNA expression levels of miR-29b and HSP47 were analyzed using qPCR. (c) HSP47 luciferase activity was measured by luciferase assay. (d) The mRNA levels of EMT-related markers were measured using qPCR. (e) Protein expression levels of HSP47, E-cadherin, α-SMA, vimentin and fibronectin were determined using Western blotting. (f) The cells were treated with TGF-β1 for 72 h after transfection with miR-29b inhibitor, and then assessed for HSP47 (1st line, green), vimentin (1st line, red), α-SMA (2nd, green), and E-cadherin (2nd, red) expression/localization using immunofluorescence. Nuclei were stained with DAPI (blue). Scale bar = 20 μm. Values are expressed as mean ± SEM of three independent samples. * p < 0.05 vs. control + miR Control; † p < 0.05, vs. TGF-β1 + miR Control.
Ijms 26 11865 g003
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MDPI and ACS Style

Shin, J.-M.; Park, J.-H.; Yang, H.-W.; Moon, J.W.; Lee, H.-M.; Park, I.-H. Correction: Shin et al. miR-29b Regulates TGF-β1-Induced Epithelial–Mesenchymal Transition by Inhibiting Heat Shock Protein 47 Expression in Airway Epithelial Cells. Int. J. Mol. Sci. 2021, 22, 11535. Int. J. Mol. Sci. 2025, 26, 11865. https://doi.org/10.3390/ijms262411865

AMA Style

Shin J-M, Park J-H, Yang H-W, Moon JW, Lee H-M, Park I-H. Correction: Shin et al. miR-29b Regulates TGF-β1-Induced Epithelial–Mesenchymal Transition by Inhibiting Heat Shock Protein 47 Expression in Airway Epithelial Cells. Int. J. Mol. Sci. 2021, 22, 11535. International Journal of Molecular Sciences. 2025; 26(24):11865. https://doi.org/10.3390/ijms262411865

Chicago/Turabian Style

Shin, Jae-Min, Joo-Hoo Park, Hyun-Woo Yang, Jee Won Moon, Heung-Man Lee, and Il-Ho Park. 2025. "Correction: Shin et al. miR-29b Regulates TGF-β1-Induced Epithelial–Mesenchymal Transition by Inhibiting Heat Shock Protein 47 Expression in Airway Epithelial Cells. Int. J. Mol. Sci. 2021, 22, 11535" International Journal of Molecular Sciences 26, no. 24: 11865. https://doi.org/10.3390/ijms262411865

APA Style

Shin, J.-M., Park, J.-H., Yang, H.-W., Moon, J. W., Lee, H.-M., & Park, I.-H. (2025). Correction: Shin et al. miR-29b Regulates TGF-β1-Induced Epithelial–Mesenchymal Transition by Inhibiting Heat Shock Protein 47 Expression in Airway Epithelial Cells. Int. J. Mol. Sci. 2021, 22, 11535. International Journal of Molecular Sciences, 26(24), 11865. https://doi.org/10.3390/ijms262411865

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