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International Journal of Molecular Sciences
  • Correction
  • Open Access

30 July 2025

Correction: Singrang et al. Melatonin Inhibits Hypoxia-Induced Alzheimer’s Disease Pathogenesis by Regulating the Amyloidogenic Pathway in Human Neuroblastoma Cells. Int. J. Mol. Sci. 2024, 25, 5225

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1
Chulabhorn Graduate Institute, Bangkok 10210, Thailand
2
Innovative Learning Center, Srinakharinwirot University, Bangkok 10110, Thailand
3
Research Center for Neuroscience, Institute of Molecular Biosciences, Mahidol University, Nakhon Pathom 73170, Thailand
*
Author to whom correspondence should be addressed.
This article belongs to the Section Molecular Neurobiology

Error in Figure

In the original publication [1], there was a mistake in Figure 1a as published. The incorrect image was inadvertently used during final preparation. The corrected Figure 1a appears below.
Ijms 26 07338 i001

Text Correction

There was an error in the original publication [1]: a correction has been made to the Methods section, in the subsection “Statistical Analysis”:
4.9. Statistical Analysis
For all the experiments, the data were analyzed, and statistical significance was determined using GraphPad Prism version 8.3.0. Differences between two groups were assessed using unpaired t tests (where equal variances were not assumed). Differences between multiple groups were assessed using a one-way analysis of variance (ANOVA), followed by Tukey’s multiple comparison test (where equal variances were not assumed). The data are expressed as the mean ± S.E.M. p values < 0.05 were considered to indicate statistical significance.
The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Singrang, N.; Nopparat, C.; Panmanee, J.; Govitrapong, P. Melatonin Inhibits Hypoxia-Induced Alzheimer’s Disease Pathogenesis by Regulating the Amyloidogenic Pathway in Human Neuroblastoma Cells. Int. J. Mol. Sci. 2024, 25, 5225. [Google Scholar] [CrossRef] [PubMed]
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