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International Journal of Molecular Sciences
  • Correction
  • Open Access

25 December 2019

Correction: Kim, Y. K. et al. Tussilagone Inhibits the Inflammatory Response and Improves Survival in CLP-Induced Septic Mice. Int. J. Mol. Sci. 2017, 18, 2744

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1
Nano-Bio Resources Center, Department of Cosmetic Sciences, Sookmyung Women’s University, Seoul 04310, Korea
2
Department of Integrative Medical Sciences, Nambu University, Gwangju 506-706, Korea
3
Department of Convergence Medicine, Asan Institute for Life Sciences, University of Ulsan College of Medicine, Asan Medical Center, 88 Olympic-ro 43-gil, Songpa-gu, Seoul 05505, Korea
4
College of Pharmacy and Natural Medicine Research Institute, Mokpo National University, Muan, Jeonnam 58554, Korea
This article belongs to the Special Issue Traditional Medicine – Unraveling Its Molecular Mechanism
We wish to make the following corrections to this paper [1]:
We found that Figure 7A,C data were unintentionally reused from the previously published data [2]. The mistake happened during the preparation of data figures for the revision in the peer-review process. All authors regret that error.
Due to the incorrect figure in original Figure 7A,C, replace the following
Ijms 21 00174 i001
with the corrected Figure 7 (Figure 1)
Figure 1. Effect of TS on survival and lung injury in cecal ligation and puncture (CLP)-induced septic mice. (A) To examine the effect of TS on the survival of CLP-induced septic mice, survival of mice was then monitored every 24 h for up to 8 days for the following experimental groups (a) sham control; mice were orally administered with either (b) vehicle (corn oil, 0.1 mL per mouse, n = 5), (c) 1 mg/kg TS (n = 5), or (d) 10 mg/kg TS (n = 5), 2 h prior to the operation. Significantly different from CLP-induced septic group (B) Expression of COX-2 and TNF-α transcripts in the isolated PAM were determined by real-time PCR; * p < 0.05 vs. CLP-induced septic group (n = 3 in each group) (C) The lungs from each experimental group were processed for histologic evaluation 1 day after CLP. Representative histologic changes in lung tissue obtained from mice belonging to each group are displayed and the arrows indicate the damaged area (hematoxylin and eosin staining; magnification 400×). Scale bar represents 200 um. (D) The extent of lung injury was estimated using scores in different sections for neutrophil infiltration, hemorrhage, necrosis, congestion, and edema. * p < 0.05 vs. CLP-induced septic group (n = 3 in each group).
We would like to apologize for any inconvenience caused to the readers by these changes.

Conflicts of Interest

The authors declare no conflict of interest.

References

  1. Kim, Y.K.; Yeo, M.G.; Oh, B.K.; Kim, H.Y.; Yang, H.J.; Cho, S.S.; Gil, M.; Lee, K.J. Tussilagone Inhibits the Inflammatory Response and Improves Survival in CLP-Induced Septic Mice. Int. J. Mol. Sci. 2017, 18, 2744. [Google Scholar] [CrossRef] [PubMed]
  2. Gil, M.; Kim, Y.K.; Hong, S.B.; Lee, K.J. Naringin Decreases TNF-α and HMGB1 Release from LPS-Stimulated Macrophages and Improves Survival in a CLP-Induced Sepsis Mice. PLoS ONE 2016, 11, e0164186. [Google Scholar] [CrossRef] [PubMed]

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