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Galectin-3 in Atrial Fibrillation: Mechanisms and Therapeutic Implications

1
Cardiology Department, Trousseau Hospital, François Rabelais University, 37000 Tours, France
2
Biochemistry Department, Trousseau Hospital, François Rabelais University, 37000 Tours, France
*
Author to whom correspondence should be addressed.
Int. J. Mol. Sci. 2018, 19(4), 976; https://doi.org/10.3390/ijms19040976
Received: 21 February 2018 / Revised: 15 March 2018 / Accepted: 23 March 2018 / Published: 25 March 2018
(This article belongs to the Special Issue Galectins in Cancer and Translational Medicine)
Maintenance of atrial fibrillation is a complex mechanism, including extensive electrical and structural remodeling of the atria which involves progressive fibrogenesis. Galectin-3 is a biomarker of fibrosis, and, thus, may be involved in atrial remodeling in atrial fibrillation patients. We review the role of galectin-3 in AF mechanisms and its potential therapeutic implications. View Full-Text
Keywords: galectin-3; atrial fibrillation; ablation galectin-3; atrial fibrillation; ablation
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MDPI and ACS Style

Clementy, N.; Piver, E.; Bisson, A.; Andre, C.; Bernard, A.; Pierre, B.; Fauchier, L.; Babuty, D. Galectin-3 in Atrial Fibrillation: Mechanisms and Therapeutic Implications. Int. J. Mol. Sci. 2018, 19, 976.

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