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Correction to Cells 2020, 9(10), 2177.
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Correction

Correction: Dallons et al. GPR91 Receptor Mediates Protection against Doxorubicin-Induced Cardiotoxicity without Altering Its Anticancer Efficacy. An In Vitro Study on H9C2 Cardiomyoblasts and Breast Cancer-Derived MCF-7 Cells. Cells 2020, 9, 2177

Department of Human Biology & Toxicology, Faculty of Medicine and Pharmacy, University of Mons, Place du Parc 20, 7000 Mons, Belgium
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Author to whom correspondence should be addressed.
Cells 2025, 14(20), 1574; https://doi.org/10.3390/cells14201574
Submission received: 23 September 2025 / Accepted: 29 September 2025 / Published: 10 October 2025
(This article belongs to the Special Issue The Molecular and Cellular Basis of Cardiovascular Disease)

Error in Figure 1

In the original publication [1], there was a mistake in Figure 1. Indirect immunofluorescence staining of the GPR91 receptor on H9C2 and MCF-7 cells. The wrong picture for the “secondary AB + DAPI” condition for H9C2 cells was used. The picture used was the same as the “primary AB + secondary AB + DAPI” condition for H9C2 cells, with a different frame and with the blue channel reading only. The corrected figure, Figure 1. Indirect immunofluorescence staining of the GPR91 receptor on H9C2 and MCF-7 cells, appears below.
The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Dallons, M.; Alpan, E.; Schepkens, C.; Tagliatti, V.; Colet, J.-M. GPR91 Receptor Mediates Protection against Doxorubicin-Induced Cardiotoxicity without Altering Its Anticancer Efficacy. An In Vitro Study on H9C2 Cardiomyoblasts and Breast Cancer-Derived MCF-7 Cells. Cells 2020, 9, 2177. [Google Scholar] [CrossRef]
Figure 1. Indirect immunofluorescence staining of the GPR91 receptor on H9C2 and MCF-7 cells. Cells were labeled with 4′,6-diamidino-2-phénylindole (DAPI) and rabbit anti-GPR91 antibody (AB) revelated with fluorescent Alexa Fluor 488 secondary AB. A control without primary AB was made for both cell lines.
Figure 1. Indirect immunofluorescence staining of the GPR91 receptor on H9C2 and MCF-7 cells. Cells were labeled with 4′,6-diamidino-2-phénylindole (DAPI) and rabbit anti-GPR91 antibody (AB) revelated with fluorescent Alexa Fluor 488 secondary AB. A control without primary AB was made for both cell lines.
Cells 14 01574 g001
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MDPI and ACS Style

Dallons, M.; Alpan, E.; Schepkens, C.; Tagliatti, V.; Colet, J.-M. Correction: Dallons et al. GPR91 Receptor Mediates Protection against Doxorubicin-Induced Cardiotoxicity without Altering Its Anticancer Efficacy. An In Vitro Study on H9C2 Cardiomyoblasts and Breast Cancer-Derived MCF-7 Cells. Cells 2020, 9, 2177. Cells 2025, 14, 1574. https://doi.org/10.3390/cells14201574

AMA Style

Dallons M, Alpan E, Schepkens C, Tagliatti V, Colet J-M. Correction: Dallons et al. GPR91 Receptor Mediates Protection against Doxorubicin-Induced Cardiotoxicity without Altering Its Anticancer Efficacy. An In Vitro Study on H9C2 Cardiomyoblasts and Breast Cancer-Derived MCF-7 Cells. Cells 2020, 9, 2177. Cells. 2025; 14(20):1574. https://doi.org/10.3390/cells14201574

Chicago/Turabian Style

Dallons, Matthieu, Esma Alpan, Corentin Schepkens, Vanessa Tagliatti, and Jean-Marie Colet. 2025. "Correction: Dallons et al. GPR91 Receptor Mediates Protection against Doxorubicin-Induced Cardiotoxicity without Altering Its Anticancer Efficacy. An In Vitro Study on H9C2 Cardiomyoblasts and Breast Cancer-Derived MCF-7 Cells. Cells 2020, 9, 2177" Cells 14, no. 20: 1574. https://doi.org/10.3390/cells14201574

APA Style

Dallons, M., Alpan, E., Schepkens, C., Tagliatti, V., & Colet, J.-M. (2025). Correction: Dallons et al. GPR91 Receptor Mediates Protection against Doxorubicin-Induced Cardiotoxicity without Altering Its Anticancer Efficacy. An In Vitro Study on H9C2 Cardiomyoblasts and Breast Cancer-Derived MCF-7 Cells. Cells 2020, 9, 2177. Cells, 14(20), 1574. https://doi.org/10.3390/cells14201574

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