Wu, Y.-J.; Su, T.-R.; Dai, G.-F.; Su, J.-H.; Liu, C.-I.
Flaccidoxide-13-Acetate-Induced Apoptosis in Human Bladder Cancer Cells is through Activation of p38/JNK, Mitochondrial Dysfunction, and Endoplasmic Reticulum Stress Regulated Pathway. Mar. Drugs 2019, 17, 287.
https://doi.org/10.3390/md17050287
AMA Style
Wu Y-J, Su T-R, Dai G-F, Su J-H, Liu C-I.
Flaccidoxide-13-Acetate-Induced Apoptosis in Human Bladder Cancer Cells is through Activation of p38/JNK, Mitochondrial Dysfunction, and Endoplasmic Reticulum Stress Regulated Pathway. Marine Drugs. 2019; 17(5):287.
https://doi.org/10.3390/md17050287
Chicago/Turabian Style
Wu, Yu-Jen, Tzu-Rong Su, Guo-Fong Dai, Jui-Hsin Su, and Chih-I Liu.
2019. "Flaccidoxide-13-Acetate-Induced Apoptosis in Human Bladder Cancer Cells is through Activation of p38/JNK, Mitochondrial Dysfunction, and Endoplasmic Reticulum Stress Regulated Pathway" Marine Drugs 17, no. 5: 287.
https://doi.org/10.3390/md17050287
APA Style
Wu, Y.-J., Su, T.-R., Dai, G.-F., Su, J.-H., & Liu, C.-I.
(2019). Flaccidoxide-13-Acetate-Induced Apoptosis in Human Bladder Cancer Cells is through Activation of p38/JNK, Mitochondrial Dysfunction, and Endoplasmic Reticulum Stress Regulated Pathway. Marine Drugs, 17(5), 287.
https://doi.org/10.3390/md17050287