Morishima, M.;                     Wang, P.;                     Horii, K.;                     Horikawa, K.;                     Ono, K.    
        Eicosapentaenoic Acid Rescues Cav1.2-L-Type Ca2+ Channel Decline Caused by Saturated Fatty Acids via Both Free Fatty Acid Receptor 4-Dependent and -Independent Pathways in Cardiomyocytes. Int. J. Mol. Sci. 2024, 25, 7570.
    https://doi.org/10.3390/ijms25147570
    AMA Style
    
                                Morishima M,                                 Wang P,                                 Horii K,                                 Horikawa K,                                 Ono K.        
                Eicosapentaenoic Acid Rescues Cav1.2-L-Type Ca2+ Channel Decline Caused by Saturated Fatty Acids via Both Free Fatty Acid Receptor 4-Dependent and -Independent Pathways in Cardiomyocytes. International Journal of Molecular Sciences. 2024; 25(14):7570.
        https://doi.org/10.3390/ijms25147570
    
    Chicago/Turabian Style
    
                                Morishima, Masaki,                                 Pu Wang,                                 Kosuke Horii,                                 Kazuki Horikawa,                                 and Katsushige Ono.        
                2024. "Eicosapentaenoic Acid Rescues Cav1.2-L-Type Ca2+ Channel Decline Caused by Saturated Fatty Acids via Both Free Fatty Acid Receptor 4-Dependent and -Independent Pathways in Cardiomyocytes" International Journal of Molecular Sciences 25, no. 14: 7570.
        https://doi.org/10.3390/ijms25147570
    
    APA Style
    
                                Morishima, M.,                                 Wang, P.,                                 Horii, K.,                                 Horikawa, K.,                                 & Ono, K.        
        
        (2024). Eicosapentaenoic Acid Rescues Cav1.2-L-Type Ca2+ Channel Decline Caused by Saturated Fatty Acids via Both Free Fatty Acid Receptor 4-Dependent and -Independent Pathways in Cardiomyocytes. International Journal of Molecular Sciences, 25(14), 7570.
        https://doi.org/10.3390/ijms25147570