Akita, S.; Suzuki, K.; Yoshimoto, H.; Ohtsuru, A.; Hirano, A.; Yamashita, S.
Cellular Mechanism Underlying Highly-Active or Antiretroviral Therapy-Induced Lipodystrophy: Atazanavir, a Protease Inhibitor, Compromises Adipogenic Conversion of Adipose-Derived Stem/Progenitor Cells through Accelerating ER Stress-Mediated Cell Death in Differentiating Adipocytes. Int. J. Mol. Sci. 2021, 22, 2114.
https://doi.org/10.3390/ijms22042114
AMA Style
Akita S, Suzuki K, Yoshimoto H, Ohtsuru A, Hirano A, Yamashita S.
Cellular Mechanism Underlying Highly-Active or Antiretroviral Therapy-Induced Lipodystrophy: Atazanavir, a Protease Inhibitor, Compromises Adipogenic Conversion of Adipose-Derived Stem/Progenitor Cells through Accelerating ER Stress-Mediated Cell Death in Differentiating Adipocytes. International Journal of Molecular Sciences. 2021; 22(4):2114.
https://doi.org/10.3390/ijms22042114
Chicago/Turabian Style
Akita, Sadanori, Keiji Suzuki, Hiroshi Yoshimoto, Akira Ohtsuru, Akiyoshi Hirano, and Shunichi Yamashita.
2021. "Cellular Mechanism Underlying Highly-Active or Antiretroviral Therapy-Induced Lipodystrophy: Atazanavir, a Protease Inhibitor, Compromises Adipogenic Conversion of Adipose-Derived Stem/Progenitor Cells through Accelerating ER Stress-Mediated Cell Death in Differentiating Adipocytes" International Journal of Molecular Sciences 22, no. 4: 2114.
https://doi.org/10.3390/ijms22042114
APA Style
Akita, S., Suzuki, K., Yoshimoto, H., Ohtsuru, A., Hirano, A., & Yamashita, S.
(2021). Cellular Mechanism Underlying Highly-Active or Antiretroviral Therapy-Induced Lipodystrophy: Atazanavir, a Protease Inhibitor, Compromises Adipogenic Conversion of Adipose-Derived Stem/Progenitor Cells through Accelerating ER Stress-Mediated Cell Death in Differentiating Adipocytes. International Journal of Molecular Sciences, 22(4), 2114.
https://doi.org/10.3390/ijms22042114