Sleep, Autonomic Nervous Function and Atherosclerosis
AbstractBehavioral and psychosocial factors related to development of cardiovascular disease have been gaining increased attention. Notably, sleep is considered to be one of the most important behavioral factors involved in progression of atherosclerosis and cardiovascular events, with autonomic nervous function a potential mechanism. Several studies have shown associations of sleep and autonomic dysfunction with major surrogate markers of atherosclerosis, such as carotid intima-media thickness and arterial stiffness. Endocrinological, immunological, oxidative, inflammatory, and metabolic responses, as well as endothelial dysfunction may mediate the effects of the autonomic nervous system. For this review, we examined recent findings related to sleep, autonomic nervous dysfunction, and atherosclerosis, with the aim of understanding the involved pathophysiological mechanisms. View Full-Text
Share & Cite This Article
Kadoya, M.; Koyama, H. Sleep, Autonomic Nervous Function and Atherosclerosis. Int. J. Mol. Sci. 2019, 20, 794.
Kadoya M, Koyama H. Sleep, Autonomic Nervous Function and Atherosclerosis. International Journal of Molecular Sciences. 2019; 20(4):794.Chicago/Turabian Style
Kadoya, Manabu; Koyama, Hidenori. 2019. "Sleep, Autonomic Nervous Function and Atherosclerosis." Int. J. Mol. Sci. 20, no. 4: 794.
Note that from the first issue of 2016, MDPI journals use article numbers instead of page numbers. See further details here.