Roles of Oral Infections in the Pathomechanism of Atherosclerosis
Abstract
:1. Introduction
2. Oral Infections: Periodontitis, Gingivitis and Endodontic Lesions
3. Potential Role of Bacteremia
4. Potential Role of Systemic Inflammation
5. Potential Role of Autoimmunity
6. Potential Role of Bacterial Toxins
7. Discussion
Acknowledgments
Conflicts of Interest
References
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Cytokine | Familiy |
---|---|
IL-8, MIP-1, MCP-1, RANTES | Chemotactic |
IL-1α, IL-1β, TNFα, IL-6, PAF | Pro-inflammatory |
IL-1RA, IL-4, IL-10 | Anti-inflammatory |
IFN-γ, IL-2, IL-4, IL-5, IL-7 | Immunoregulatory |
PDGF, EGF, FGF, IGF, VEGF | Growth factor |
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Aarabi, G.; Heydecke, G.; Seedorf, U. Roles of Oral Infections in the Pathomechanism of Atherosclerosis. Int. J. Mol. Sci. 2018, 19, 1978. https://doi.org/10.3390/ijms19071978
Aarabi G, Heydecke G, Seedorf U. Roles of Oral Infections in the Pathomechanism of Atherosclerosis. International Journal of Molecular Sciences. 2018; 19(7):1978. https://doi.org/10.3390/ijms19071978
Chicago/Turabian StyleAarabi, Ghazal, Guido Heydecke, and Udo Seedorf. 2018. "Roles of Oral Infections in the Pathomechanism of Atherosclerosis" International Journal of Molecular Sciences 19, no. 7: 1978. https://doi.org/10.3390/ijms19071978