Mechanisms of Cardiac Arrhythmias: Focus on Cardiomyocytes
A special issue of Journal of Cardiovascular Development and Disease (ISSN 2308-3425). This special issue belongs to the section "Electrophysiology and Cardiovascular Physiology".
Deadline for manuscript submissions: closed (20 February 2024) | Viewed by 379
Special Issue Editor
Interests: electrophysiology; hypertension; arrhythmias; echocardiography; cardiomyocytes; clinical cardiology; cardiomyocyte calcium handling; mitochondrial function
Special Issue Information
Dear Colleagues,
Cardiac arrhythmias are the result of complex interactions between various factors that affect the electrical activity of cardiomyocytes. These factors include ion channel dysfunction, dysregulation of calcium handling, myocardial ischemia, structural abnormalities in the heart, imbalances in the autonomic nervous system, and genetic mutations. Understanding these mechanisms is crucial for the development of effective treatments for arrhythmias. In cardiomyocytes, alterations in ion channels and calcium handling can lead to abnormal electrical activity, while structural abnormalities and ischemia can alter the conduction of electrical impulses. The autonomic nervous system also plays an important role in regulating heart rate and rhythm. Inherited genetic mutations can cause ion channel dysfunction or structural abnormalities in the heart, leading to arrhythmias.
Overall, a comprehensive understanding of the mechanisms of cardiac arrhythmias is essential for the development of effective therapies to prevent and treat these potentially life-threatening conditions.
Dr. Marie-Louise Ward
Guest Editor
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Keywords
- arrhythmogenic cardiomyopathies
- diastolic Ca2+ leak
- inflammatory cytokines
- impaired Ca2+ cycling
- cardiac channelopathies
- mitochondrial dysfunction
- membrane potential
- myocardial ischemia
- reactive oxygen species
- t-tubule disorganization
- unbalanced membrane currents
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