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Editorial

Muscles—A New Open Access Journal

Neuromuscular Lab, Department of Neurosciences, University of Padova, Campus Biomedico Pietro D’Abano, Via Giuseppe Orus 2B, 35131 Padova, Italy
Muscles 2022, 1(1), 48-49; https://doi.org/10.3390/muscles1010004
Submission received: 28 February 2022 / Accepted: 1 March 2022 / Published: 9 March 2022
Skeletal muscle represents over 40% of the body and is important for metabolism, exercise, and movement. The acute nature of muscle energy failure is manifested in a metabolic crisis with muscle weakness, sometimes associated with acute failure, while exercise is vital for muscle maintenance and regeneration.
In muscle, several organelles (sarcoplasmic reticulum, triads, T-tubules, mitochondria, etc.) contribute to the regulation of calcium homeostasis, oxidative stress, mitochondrial function, and diminished activity of the potent anabolic regulator mTOR (mammalian target of rapamycin), including its failure to shuttle to and from the lysosomes in response to cellular stress. All these organelles may contribute to tissue damage in aging and muscle diseases. Autophagy is controlled by FoxO transcription factors that control protein expression [1].
Why do we need a muscle journal? An easy answer would simply be to say that this journal will be indeed needed for the recent progress in the field, particularly sections that will be devoted to muscle biochemistry, biology, and pharmacology; however, the response is simply that there is currently an explosion of new techniques and approaches in the field of muscle biology and pathology, where new techniques such as next generation sequencing and muscle imaging are needed for its full interpretation and discussion. This was the impetus for starting the journal in which you are now collecting both human and animal models of muscle adaptation and plasticity, as well as the pathophysiology of muscle disease.
I am pleased to announce a new open access journal—Muscles (ISSN: 2813-0413) [2]. Muscles will provide a platform for scientists and academics all over the world to promote, share, and discuss various new advances and developments in the field of muscles.
Muscles will publish papers covering a broad range of areas, including but not limited to: muscle microanatomy; muscle biochemistry; muscle cell biology; muscle epidemiology; muscle immunology; muscle pathology; muscle pharmacology; muscle physiology; muscle toxicology, muscle diseases, etc.
We welcome you to Muscles and invite you to contribute your papers or submit Special Issue proposals. We look forward to receiving your manuscripts for publication in this journal.

Conflicts of Interest

The author declares no conflict of interest.

References

  1. Nascimbeni, A.C.; Fanin, M.; Masiero, E.; Angelini, C.; Sandri, M. Impaired Autophagy Contributes to Muscle Atrophy in Glycogen Storage Disease Type II Patients. Autophagy 2012, 8, 1697–1700. [Google Scholar] [CrossRef] [PubMed] [Green Version]
  2. Muscles Home Page. Available online: https://www.mdpi.com/journal/muscles (accessed on 6 January 2022).

Short Biography of Author

Muscles 01 00004 i001Corrado Angelini is a full professor of neurology at the University of Padova, Italy, where he also served as coordinator of the PhD program in neurosciences, and director of the Neurology Residency Program and the Department of Neurosciences Neuromuscular Center. Dr. Angelini earned his medical degree from the University of Padova and completed two fellowships with the Muscular Dystrophy Association, a research fellowship in 1972 at the Mayo Clinic, and a senior fellowship in 1978 at the University of California, Los Angeles. In 1978–1979, he was a visiting assistant professor of neurology at the Reed Neurological Research Center, UCLA Medical School. In 1984, he was a visiting professor at the University of Colorado, Denver and the University of Boston. His major research interests are primary biochemical defects in inherited neuromuscular diseases, clinical trials, muscle dystrophies, FSHD, LGMD, BMD, DMD, ALS, myasthenia gravis, congenital muscle diseases, carnitine and lipid metabolism, glycogenosis, biomarkers in metabolic and mitochondrial disorders, hypoxia, and high-altitude effect in neurological disorders.
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MDPI and ACS Style

Angelini, C. Muscles—A New Open Access Journal. Muscles 2022, 1, 48-49. https://doi.org/10.3390/muscles1010004

AMA Style

Angelini C. Muscles—A New Open Access Journal. Muscles. 2022; 1(1):48-49. https://doi.org/10.3390/muscles1010004

Chicago/Turabian Style

Angelini, Corrado. 2022. "Muscles—A New Open Access Journal" Muscles 1, no. 1: 48-49. https://doi.org/10.3390/muscles1010004

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