Abstract
In this paper, we introduce a fast iterative scheme and establish its convergence under a contractive condition. This new scheme can be viewed as an extension and generalization of existing iterative schemes such as Picard–Noor and UO iterative schemes for solving nonlinear equations. We demonstrate theoretically and numerically that the new scheme converges faster than several existing iterative schemes with the fastest known convergence rates for contractive mappings. We also analyze the stability of the new scheme and provide numerical computations to validate the analytic results. Finally, we implement the new scheme in MATLAB R2023b to simulate the dynamics of the Ebola virus disease.
Keywords:
fixed point iterative scheme; rate of convergence; fixed point approximation; stability; data dependence; Ebola epidemic model MSC:
46A03; 47H09; 47H10; 47J26; 45D05; 34A08
1. Introduction
1.1. Background and Motivation
One of the most efficient ways of solving nonlinear problems of the form
is to reduce them to their equivalent fixed point problems of the form
where is a suitable mapping. The solution of (2), often obtained as the limit of an iterative sequence, unlocks the corresponding solution of (1). Classical schemes such as Picard [1], Mann [2], and Ishikawa [3] have long been used in finding such limits.
While these classical methods are easy to implement, they often suffer from slow convergence, which limits their practical efficiency for solving real-world nonlinear problems. To overcome the limitations of classical schemes, several modified and hybrid iterations have been proposed.
1.2. Some Modified Iterative Schemes
Modified iterative schemes are developed to solve nonlinear equations, integral equations, and optimization problems more efficiently. These modifications often introduce control parameters (e.g., step sizes) or combine multiple iterates to improve convergence rates.
For instance, Noor [4] proposed a three-step iterative scheme generalizing Mann and Ishikawa iterative schemes. The normal S-iterative scheme, introduced in [5,6], is the hybrid of Picard and Mann iterative schemes and has been applied to a mixed-type Volterra--Fredholm functional nonlinear integral equation [7].
However, many existing schemes show different convergence behaviors depending on the problem class and contractive condition [8]. For instance, Berinde [9] showed that the Picard iteration converges faster than the Mann iteration in the class of Zamfirescu operators. In [6], the S-iterative scheme introduced by Agarwal et al. [10] was shown to converge faster than the Picard iterative scheme for contraction mappings. Khan [5] showed that the normal S-iterative scheme converges faster than all of the Picard, Mann and Ishikawa iterative schemes for contraction mappings. The modified SP iterative scheme developed in [11] converges faster than the normal S-iterative scheme. More recently, the Picard–Noor (3) and UO (4) iterative schemes have been developed to further accelerate convergence [12,13].
1.3. Research Gap and Objective
Despite recent progress, the search for iterative schemes with convergent rates surpassing those of existing leading schemes continues. This motivates the central research question of this paper:
Is there an iterative scheme with a better convergence rate than the Picard–Noor and UO iterative schemes under contractive mappings?
This paper aims to address this question by introducing a new scheme designed to improve the convergence rate under contractive conditions.
1.4. Proposed Iterative Scheme
Let be a nonempty convex subset of a Banach space , be a contraction mapping, and the set of all fixed points of . Let be an initial guess. The control sequences are assumed to be constant unless otherwise stated. The new scheme defined below generates a sequence that converges to a fixed point of . Each step uses the previous approximation to produce a better one.
The rest of the paper is structured as follows: we prove the convergence, stability, and data dependence results of the proposed iterative scheme; compare its rate of convergence with existing schemes; provide numerical validation; and apply the method to simulate the dynamics of an Ebola epidemic model.
2. Convergence Theorem
To analyze the convergence behavior of our new scheme, we recall an important defiinition concerning contractive mappings, which will be instrumental in establishing our result.
Definition 1.
A contraction mapping satisfies the following equation,
for all , and .
We prove the convergence of the new scheme under a contraction mapping.
Theorem 1.
Assume that is a nonempty closed convex subset of a Banach space and be a contraction maping satisfying Definition 1 such that . Let be an iterative sequence generated by the new scheme (1) with real sequences satisfying
Then, converges to a unique fixed point of , say .
Proof.
From the Banach contraction principle [14], the existence and uniqueness of is guaranteed. It remains to show that .
Using Definition 1, and the new scheme in Algorithm 1, we have that
since , , and .
Remark 1.
Theorem 1 shows that the new scheme converges to the unique fixed point of contractive mappings.
In the following section, we establish the data dependence and stability results for the new scheme in Algorithm 1.
| Algorithm 1 Fast fixed point iterative scheme |
Set a tolerance for do if then break end if end for |
3. Data Dependence and Stability Results
The following definition and lemmas, adapted from the existing literature, are necessary to establish the data dependency and stability results.
Definition 2
([15,16]). Let and define the iterative scheme which produces a sequence in . Suppose that converges strongly to , where denotes the set of all fixed points of . Assume that is an arbitrary bounded sequence in and set
The iterative scheme is said to be stable if and only if implies that .
Lemma 1
([17]). If ρ is a real number such that , and is a sequence of positive numbers such that , then for any sequence of positive numbers satisfying
we have that
Lemma 2
([16]). Assume there exists for the non-negative real sequences such that for all ,
where , , and , for all , then
Theorem 2.
Let be an approximate operator of a contraction mapping , and be an approximate of the iterative sequence generated by the new scheme in Algorithm 1. Define as follows:
where the real sequences , , satisfy the condition: . If and such that , then we have that where is a constant.
Proof.
Since , , , (26),
Let , and . From Lemma 2, we have that . Again, from Theorem 1, we can confirm that . Thus, given , we have that . □
Finally, we show that the new scheme in Algorithm 1 is -stable.
Theorem 3.
Let , and be as defined in Theorem 1 such that and is the unique fixed point of . Let be a sequence generated by the new scheme, as detailed in Algorithm 1, which converges to . Then, the new scheme is -stable.
Proof.
Let be an arbitrary sequence in and let the sequence generated by the new scheme in Algorithm 1 converge to . Let . We want to show that if and only if .
Set . Suppose and using (11),
So that from Lemma 1, .
Conversely, suppose that , then using the result from (11)
We have that . Combining the two cases, Definition 2 is satisfied. Thus, the new scheme is -stable. □
Remark 2.
Since a -stable iterative scheme is also almost -stable, but not vice versa [15], we conclude that the new scheme is also almost -stable.
4. Rate of Convergence
The Picard–Noor (3) and UO iterative schemes (4) have some of the fastest known convergence rates for contractive mappings. Therefore, they provide a strong benchmark for evaluating the efficiency of our new scheme. Our goal is to demonstrate that the new scheme is not only convergent but also superior in terms of convergence rate when compared with leading schemes.
Definition 3
([9]). Let the two real sequences and converge to μ and ν, respectively, and assume there exists
If , then it can be said that converges faster to μ than to ν.
Theorem 4.
Let be a nonempty closed convex subset of a Banach space and be a contraction maping satisfying the contractive condition in Definition 1 and having a fixed point . Let be real sequences for . Consider the iterative sequences , , and defined by the Picard–Noor, the UO, and our new scheme, respectively. Then, converges faster to the fixed point than and .
Proof.
Theorem 4 establishes that the new scheme converges faster to the fixed point of contraction mappings than the Picard–Noor and the UO iterative schemes. Thus, the new scheme is an improvement in terms of convergence speed compared to the existing scheme. We now support our analytic results using some numerical examples.
5. Numerical Computations
Example 1.
Let , and consider the following affine transformation, defined by
for all . It is easy to see that is a contraction map with contractive constant , since
for all . Let for each with the initial value . The set of fixed points of is .
Table 1 compares the number of iterations required by the new scheme and the two benchmark schemes—UO and Picard–Noor—to converge to the fixed point, . It is evident from the table that the new scheme requires the fewest number of steps to attain the fixed point up to a specified tolerance, indicating its computational advantage.
Table 1.
A comparison of our scheme with Picard–Noor and UO schemes.
Figure 1 provides a visual comparison of the convergence behavior of the three schemes. The plot shows the rapid decay of the iterates produced by the new scheme, validating its superior convergence rate.
Figure 1.
Rate of convergence for iterative schemes.
Figure 2 displays the residual norms at each step for all schemes. The residual represents the deviation between the image of the current iterate under the mapping and the iterate itself. The residual norm for the new scheme decreases more rapidly than for the other two schemes. This implies that for each iteration, the new method yields a better approximation of the fixed point. Such behavior is valuable in practical problems where reducing the number of iterations translates to saving computational resources. Furthermore, we consider Example 1 under slight perturbations of the initial guess. Specifically, we introduce perturbations of magnitude , , , and to the initial point and observe the behavior of the resulting sequences. Figure 3 shows that despite the initial deviations, all perturbed sequences converge rapidly to the same fixed point as the unperturbed sequence. This indicates that the effect of the perturbations diminishes progressively with each iteration, confirming the stability of the proposed scheme.
Figure 2.
Convergence behavior via residual norm for iterative schemes.
Figure 3.
Stability analysis under initial perturbations.
Example 2.
Consider the nonlinear mapping
for all .
We show that on the interval is a contraction mapping with a Lipschitz constant, . From the mean-value theorem, for any differentiable function , , for some k between x and y:
since is increasing on , so that
Therefore, is a contraction mapping on . The Banach contraction principle guarantees that there exists a unique fixed point, . Let for each and the initial point . We now compare the convergence rates of the new scheme, the UO, and the Picard–Noor.
Table 2 provides numerical evidence supporting the superior convergence behavior of the new scheme when applied to a nonlinear fixed point problem. We consider the contraction mapping defined on the interval , which possesses a unique fixed point at . The table clearly demonstrates that the new scheme achieves convergence in fewer iteration steps compared to both the Picard–Noor and UO schemes in the context of nonlinear mappings.
Table 2.
A comparison of our scheme with Picard–Noor and UO schemes.
These examples demonstrate that the new scheme not only improves upon the convergence properties of existing schemes but also exhibits enhanced stability characteristics. Thus, the new scheme is well suited for applications involving uncertainty or imprecise initial data.
Having established the analytic results for the new scheme shown in Algorithm 1, we now apply the new scheme to simulate the dynamics of Ebola disease.
6. Application to Ebola Virus Disease
6.1. Fixed Point Theory in Epidemiology
Fixed point theory is a fundamental mathematical concept with wide-ranging applications [16]. In epidemiology, it is used to establish the existence and uniqueness of solutions, analyze stability, and numerically solve models [18]. ranging from proving the existence and uniqueness of solutions [19,20], to conducting stability analyses [21], and solving models numerically to simulate disease dynamics over time. For instance, in [20], fixed point theory was used to establish the existence and uniqueness of novel fractal–fractional models for Q fever transmission under the Atangana–Baleanu (Mittag–Leffler kernel) fractal–fractional operator. Similarly, in [21], the solution and stability criteria for a fractional-order (FO) HIV/AIDS model involving the Liouville–Caputo and Atangana–Baleanu–Caputo derivatives were derived using fixed point theory. In [22], fixed point theory was used to determine the optimal final time needed to reduce the number of infected people in an epidemic model with four compartments, namely classes of susceptible, controlled, infected and removed people.
6.2. Model Structure
Ebola virus disease is a severe viral illness transmitted to humans from wild animals such as fruit bats and infected individuals who are still alive or from dead to the living during funerals. It also spreads among humans primarily through direct contact with the blood, secretions, organs, or other body fluids of infected individuals as well as with surfaces and materials (such as clothing or bedding) contaminated with these fluids [23,24]. It takes about 2 to 21 days from infection to the appearance of symptoms.
We consider a delayed epidemic model with four compartments: susceptible (), exposed (), infected (), and recovered () adopted from [25,26]. The SEIR model of the Ebola virus consists of coupled nonlinear delay differential equations that track how individuals move between compartments:
with the initial conditions , , , and , where the parameters are defined in Table 3.
Table 3.
Parameter values used in the iterative algorithm and epidemic model adapted from [25,26].
The model includes a constant delay r representing the incubation period of the disease:
- Time delay effects: states at time t depend on values at ;
- Integral terms: history-dependent accumulation of infection and recovery;
- Nonlinear interaction terms involving , , and .
Our goal is to use the new scheme to solve numerically and simulate the dynamics of the Ebola virus model.
6.3. Fixed Point Reformulation
To be able to apply a fixed point scheme, the disease model is first reformulated as a fixed point problem. For each compartment, , , , , the system of Equation (53) is reformulated into integral equations and solved iteratively using the new scheme:
Integrating from , we have
which can be where is constant. , , and are reformulated in a similar way.
We apply the new scheme in Algorithm 1:
where are relaxation parameters. The simulation domain is discretized uniformly with time step . Let for such that .
A direct closed-form integral is not available because , , , and are unknown and inside a nonlinear integral with delay. We approximate the integral part using the five-point Gauss–Legendre quadrature:
where are Gauss nodes and are corresponding weights for . Since delay r may not align with discrete grid points, we estimate , , and using linear interpolation:
where .
To ensure biological realism, all compartments are projected onto the non-negative space at each step:
and the iterative scheme stops when the infinity norm of the change across iterations satisfies the following:
The purpose of each step is presented in Table 4.
Table 4.
Summary of steps.
6.4. Dynamics of the Ebola Virus Model
We apply the new scheme in Algorithm 1 to solve numerically the dynamics of Ebola spread. The steps summarized in Algorithm 2 are implemented into MATLAB R2023b.
6.5. Analysis and Discussion of the Model Dynamics
The simulation results presented in Figure 4 show the time evolution of the four compartments in the SEIR model for Ebola: susceptible , exposed , infected , and recovered populations over a 40-day period.
Figure 4.
Dynamics of the Ebola disease model.
- Susceptible Population Dynamics
Initially, the susceptible population remains constant at the initial value, indicating no immediate infection or death. After a short delay, the susceptible population experiences a sharp decline as susceptible individuals are exposed to the virus and move into the exposed class. This rapid drop reflects a high transmission rate in parameters such as the death rate of the susceptible population. Unlike in classical closed SEIR models where the total population remains constant, here, continues to decline even after the majority of susceptibles are depleted. This is because the model includes a very high death rate , which causes susceptible individuals to die over time, and the rate at which susceptible class is recruited is negligible . This means the population is open and shrinking over time.
- Exposed Population Dynamics
The exposed compartment starts from a low initial value and increases as susceptible individuals become infected but are not yet infectious. The rise of corresponds to the latent period of the disease, where individuals are incubating the virus. The exposed population reaches a peak, after which it declines as exposed individuals progress to the infected stage.
- Infected Population Dynamics
The infected population shows the typical epidemic curve with a rise following the increase in the exposed class and a peak indicating the maximum number of infectious individuals at the epidemic’s height. The peak is followed by a decline, corresponding to individuals recovering or dying.
- Recovered Population Dynamics
The recovered population initially grows as infected individuals recover. However, unlike closed SEIR models where recovered individuals accumulate indefinitely, here peaks and then declines over time.
| Algorithm 2 Steps employed in solving the Ebola virus model |
|
- Possible Improvements
The model currently assumes constant parameters (adopted from [25,26]) such as transmission and progression rates, which may not accurately reflect variations observed in outbreaks due to changes in public health interventions, viral mutations, and population behavior. Additionally, the absence of demographic processes such as birth and natural death rates limits the model’s long-term predictive capacity. Future improvements could include age-structured compartments, spatial heterogeneity, and the incorporation of vaccination and treatment effects. Such enhancements would provide a more realistic and adaptable framework to better inform public health strategies and outbreak response planning.
7. Conclusions
The development of a novel fixed point iterative scheme with a faster convergence rate contributes significantly to fixed point theory by offering a more efficient method for approximating solutions to nonlinear problems. In this paper, we introduced a fast iterative scheme which generalizes existing schemes. We presented the analysis regarding the convergence behavior, stability, and sensitivity to data. Numerical examples, illustrated through graphs and tables, further demonstrated the effectiveness and stability of the new scheme. Finally, we implemented the new scheme in MATLAB R2023b to simulate Ebola virus disease dynamics.
Author Contributions
Conceptualization, G.A.O. and E.H.A.; Methodology, G.A.O. and E.H.A.; Software, R.T.A. and E.H.A.; Validation, G.A.O. and R.T.A.; Formal analysis, G.A.O., R.T.A. and E.H.A.; Investigation, G.A.O., R.T.A. and E.H.A.; Resources, R.T.A.; Writing—original draft, E.H.A.; Writing—review & editing, G.A.O., R.T.A. and E.H.A.; Visualization, R.T.A.; Project administration, R.T.A.; Funding acquisition, R.T.A. All authors have read and agreed to the published version of the manuscript.
Funding
This work was supported and funded by the Deanship of Scientific Research at Imam Mohammad Ibn Saud Islamic University (IMSIU) (grant number IMSIU-DDRSP2502).
Data Availability Statement
The original contributions presented in this study are included in the article. Further inquiries can be directed to the corresponding author.
Acknowledgments
This work was supported and funded by the Deanship of Scientific Research at Imam Mohammad Ibn Saud Islamic University (IMSIU) (grant number IMSIU-DDRSP2502).
Conflicts of Interest
The authors declare no conflicts of interest.
Correction Statement
This article has been republished with a minor correction to the existing affiliation information. This change does not affect the scientific content of the article.
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