Colemonts-Vroninks, H.; Neuckermans, J.; Marcelis, L.; Claes, P.; Branson, S.; Casimir, G.; Goyens, P.; Martens, G.A.; Vanhaecke, T.; De Kock, J.
Oxidative Stress, Glutathione Metabolism, and Liver Regeneration Pathways Are Activated in Hereditary Tyrosinemia Type 1 Mice upon Short-Term Nitisinone Discontinuation. Genes 2021, 12, 3.
https://doi.org/10.3390/genes12010003
AMA Style
Colemonts-Vroninks H, Neuckermans J, Marcelis L, Claes P, Branson S, Casimir G, Goyens P, Martens GA, Vanhaecke T, De Kock J.
Oxidative Stress, Glutathione Metabolism, and Liver Regeneration Pathways Are Activated in Hereditary Tyrosinemia Type 1 Mice upon Short-Term Nitisinone Discontinuation. Genes. 2021; 12(1):3.
https://doi.org/10.3390/genes12010003
Chicago/Turabian Style
Colemonts-Vroninks, Haaike, Jessie Neuckermans, Lionel Marcelis, Paul Claes, Steven Branson, Georges Casimir, Philippe Goyens, Geert A. Martens, Tamara Vanhaecke, and Joery De Kock.
2021. "Oxidative Stress, Glutathione Metabolism, and Liver Regeneration Pathways Are Activated in Hereditary Tyrosinemia Type 1 Mice upon Short-Term Nitisinone Discontinuation" Genes 12, no. 1: 3.
https://doi.org/10.3390/genes12010003
APA Style
Colemonts-Vroninks, H., Neuckermans, J., Marcelis, L., Claes, P., Branson, S., Casimir, G., Goyens, P., Martens, G. A., Vanhaecke, T., & De Kock, J.
(2021). Oxidative Stress, Glutathione Metabolism, and Liver Regeneration Pathways Are Activated in Hereditary Tyrosinemia Type 1 Mice upon Short-Term Nitisinone Discontinuation. Genes, 12(1), 3.
https://doi.org/10.3390/genes12010003