Deng, K.; Li, W.; Liang, J.; Chen, Z.; Xu, Y.; Zhang, J.; Zhan, Y.; Yang, Z.; Chen, S.; Zhao, Y.-T.;
et al. Peptide DFCPPGFNTK Mitigates Dry Eye Pathophysiology by Suppressing Oxidative Stress, Apoptosis, Inflammation, and Autophagy: Evidence from In Vitro and In Vivo Models. Curr. Issues Mol. Biol. 2025, 47, 441.
https://doi.org/10.3390/cimb47060441
AMA Style
Deng K, Li W, Liang J, Chen Z, Xu Y, Zhang J, Zhan Y, Yang Z, Chen S, Zhao Y-T,
et al. Peptide DFCPPGFNTK Mitigates Dry Eye Pathophysiology by Suppressing Oxidative Stress, Apoptosis, Inflammation, and Autophagy: Evidence from In Vitro and In Vivo Models. Current Issues in Molecular Biology. 2025; 47(6):441.
https://doi.org/10.3390/cimb47060441
Chicago/Turabian Style
Deng, Kaishu, Wenan Li, Jinyuan Liang, Zhengdao Chen, Yan Xu, Jingxi Zhang, Yingtong Zhan, Zhiyou Yang, Shaohong Chen, Yun-Tao Zhao,
and et al. 2025. "Peptide DFCPPGFNTK Mitigates Dry Eye Pathophysiology by Suppressing Oxidative Stress, Apoptosis, Inflammation, and Autophagy: Evidence from In Vitro and In Vivo Models" Current Issues in Molecular Biology 47, no. 6: 441.
https://doi.org/10.3390/cimb47060441
APA Style
Deng, K., Li, W., Liang, J., Chen, Z., Xu, Y., Zhang, J., Zhan, Y., Yang, Z., Chen, S., Zhao, Y.-T., & Hu, C.
(2025). Peptide DFCPPGFNTK Mitigates Dry Eye Pathophysiology by Suppressing Oxidative Stress, Apoptosis, Inflammation, and Autophagy: Evidence from In Vitro and In Vivo Models. Current Issues in Molecular Biology, 47(6), 441.
https://doi.org/10.3390/cimb47060441
