Next Article in Journal
The Lysine at Position 177 Is Essential to Limit the Inhibitory Capacities of Sprouty4 Protein in Normal and Cancer-Derived Cells
Previous Article in Journal
Biomolecular Aspects of Reelin in Neurodegenerative Disorders: An Old Candidate for a New Linkage of the Gut–Brain–Eye Axis
 
 
Font Type:
Arial Georgia Verdana
Font Size:
Aa Aa Aa
Line Spacing:
Column Width:
Background:
Correction

Correction: Yun et al. Rhusflavone Modulates Osteoclastogenesis Through RANKL-Induced AKT Signaling in Bone Marrow-Derived Macrophages. Int. J. Mol. Sci. 2025, 26, 3025

1
Department of Oral and Maxillofacial Pathology, School of Dentistry, Kyung Hee University, Seoul 02447, Republic of Korea
2
National Institute for Korean Medicine Development, Gyeongsan 38540, Republic of Korea
3
Honam Regional Center, Korea Basic Science Institute (KBSI), Gwangju 61751, Republic of Korea
*
Authors to whom correspondence should be addressed.
Int. J. Mol. Sci. 2025, 26(15), 7354; https://doi.org/10.3390/ijms26157354
Submission received: 11 July 2025 / Accepted: 27 July 2025 / Published: 30 July 2025
(This article belongs to the Section Molecular Endocrinology and Metabolism)
In the original publication [1], there was a mistake in Figure 5B as published. The first image in Figure 5B was processed incorrectly, including the scale bar and included in error. The corrected Figure 5B appears below. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Yun, H.-M.; Kim, B.; Kim, E.; Park, K.-R. Rhusflavone Modulates Osteoclastogenesis Through RANKL-Induced AKT Signaling in Bone Marrow-Derived Macrophages. Int. J. Mol. Sci. 2025, 26, 3025. [Google Scholar] [CrossRef] [PubMed]
Figure 5. Effects of Rhus on the inhibition of receptor activator of nuclear factor kappa B ligand (RANKL)-induced AKT signaling pathway in osteoclastogenesis. (A) Bone marrow macrophages (BMMs) were differentiated with 10 μM Rhus, in the presence or absence of 1 μM SC79 or LY294002, for 1 day. Western blot analysis was used to assess c-Fos and β-actin expression levels. The relative level (%) is presented as a bar graph. (B,C) The BMMs were cultured in 30 ng/mL M-CSF and 100 ng/mL RANKL with 10 μM Rhus, in the presence or absence of 1 μM SC79 or LY294002, for 5 days. Mature osteoclasts were detected with tartrate-resistant acid phosphatase (TRAP) staining (pink) (B). TRAP-positive multinucleated osteoclasts (MNCs) were counted under a microscope (C). A quantity of 3–10 nuclei (left), 10 < nuclei (right). Scale bar: 100 μm. *, p < 0.05 compared with RANKL alone. #, p < 0.05 compared with RANKL + Rhus. The data presented are derived from three separate experiments and are expressed as the mean ± standard deviation (SD).
Figure 5. Effects of Rhus on the inhibition of receptor activator of nuclear factor kappa B ligand (RANKL)-induced AKT signaling pathway in osteoclastogenesis. (A) Bone marrow macrophages (BMMs) were differentiated with 10 μM Rhus, in the presence or absence of 1 μM SC79 or LY294002, for 1 day. Western blot analysis was used to assess c-Fos and β-actin expression levels. The relative level (%) is presented as a bar graph. (B,C) The BMMs were cultured in 30 ng/mL M-CSF and 100 ng/mL RANKL with 10 μM Rhus, in the presence or absence of 1 μM SC79 or LY294002, for 5 days. Mature osteoclasts were detected with tartrate-resistant acid phosphatase (TRAP) staining (pink) (B). TRAP-positive multinucleated osteoclasts (MNCs) were counted under a microscope (C). A quantity of 3–10 nuclei (left), 10 < nuclei (right). Scale bar: 100 μm. *, p < 0.05 compared with RANKL alone. #, p < 0.05 compared with RANKL + Rhus. The data presented are derived from three separate experiments and are expressed as the mean ± standard deviation (SD).
Ijms 26 07354 g005
Disclaimer/Publisher’s Note: The statements, opinions and data contained in all publications are solely those of the individual author(s) and contributor(s) and not of MDPI and/or the editor(s). MDPI and/or the editor(s) disclaim responsibility for any injury to people or property resulting from any ideas, methods, instructions or products referred to in the content.

Share and Cite

MDPI and ACS Style

Yun, H.-M.; Kim, B.; Kim, E.; Park, K.-R. Correction: Yun et al. Rhusflavone Modulates Osteoclastogenesis Through RANKL-Induced AKT Signaling in Bone Marrow-Derived Macrophages. Int. J. Mol. Sci. 2025, 26, 3025. Int. J. Mol. Sci. 2025, 26, 7354. https://doi.org/10.3390/ijms26157354

AMA Style

Yun H-M, Kim B, Kim E, Park K-R. Correction: Yun et al. Rhusflavone Modulates Osteoclastogenesis Through RANKL-Induced AKT Signaling in Bone Marrow-Derived Macrophages. Int. J. Mol. Sci. 2025, 26, 3025. International Journal of Molecular Sciences. 2025; 26(15):7354. https://doi.org/10.3390/ijms26157354

Chicago/Turabian Style

Yun, Hyung-Mun, Bomi Kim, Eonmi Kim, and Kyung-Ran Park. 2025. "Correction: Yun et al. Rhusflavone Modulates Osteoclastogenesis Through RANKL-Induced AKT Signaling in Bone Marrow-Derived Macrophages. Int. J. Mol. Sci. 2025, 26, 3025" International Journal of Molecular Sciences 26, no. 15: 7354. https://doi.org/10.3390/ijms26157354

APA Style

Yun, H.-M., Kim, B., Kim, E., & Park, K.-R. (2025). Correction: Yun et al. Rhusflavone Modulates Osteoclastogenesis Through RANKL-Induced AKT Signaling in Bone Marrow-Derived Macrophages. Int. J. Mol. Sci. 2025, 26, 3025. International Journal of Molecular Sciences, 26(15), 7354. https://doi.org/10.3390/ijms26157354

Note that from the first issue of 2016, this journal uses article numbers instead of page numbers. See further details here.

Article Metrics

Back to TopTop