Abdelsalam, S.S.;                     Zahid, M.A.;                     Ghanem, S.K.;                     Khan, A.;                     Parray, A.;                     Agouni, A.    
        Sestrin2 Suppression Promotes Endothelial–Mesenchymal Transition and Exacerbates Methylglyoxal-Induced Endothelial Dysfunction. Int. J. Mol. Sci. 2024, 25, 13463.
    https://doi.org/10.3390/ijms252413463
    AMA Style
    
                                Abdelsalam SS,                                 Zahid MA,                                 Ghanem SK,                                 Khan A,                                 Parray A,                                 Agouni A.        
                Sestrin2 Suppression Promotes Endothelial–Mesenchymal Transition and Exacerbates Methylglyoxal-Induced Endothelial Dysfunction. International Journal of Molecular Sciences. 2024; 25(24):13463.
        https://doi.org/10.3390/ijms252413463
    
    Chicago/Turabian Style
    
                                Abdelsalam, Shahenda Salah,                                 Muhammad Ammar Zahid,                                 Sarah Khalaf Ghanem,                                 Abbas Khan,                                 Aijaz Parray,                                 and Abdelali Agouni.        
                2024. "Sestrin2 Suppression Promotes Endothelial–Mesenchymal Transition and Exacerbates Methylglyoxal-Induced Endothelial Dysfunction" International Journal of Molecular Sciences 25, no. 24: 13463.
        https://doi.org/10.3390/ijms252413463
    
    APA Style
    
                                Abdelsalam, S. S.,                                 Zahid, M. A.,                                 Ghanem, S. K.,                                 Khan, A.,                                 Parray, A.,                                 & Agouni, A.        
        
        (2024). Sestrin2 Suppression Promotes Endothelial–Mesenchymal Transition and Exacerbates Methylglyoxal-Induced Endothelial Dysfunction. International Journal of Molecular Sciences, 25(24), 13463.
        https://doi.org/10.3390/ijms252413463