Next Article in Journal
Effect of Multiple Sclerosis Cerebrospinal Fluid and Oligodendroglia Cell Line Environment on Human Wharton’s Jelly Mesenchymal Stem Cells Secretome
Next Article in Special Issue
Current Studies of the Effects of Drought Stress on Root Exudates and Rhizosphere Microbiomes of Crop Plant Species
Previous Article in Journal
CD44 Expression Intensity Marks Colorectal Cancer Cell Subpopulations with Different Extracellular Vesicle Release Capacity
Previous Article in Special Issue
Genome-Wide Identification and Functions against Tomato Spotted Wilt Tospovirus of PR-10 in Solanum lycopersicum
 
 
Article
Peer-Review Record

Interplay between Ca2+/Calmodulin-Mediated Signaling and AtSR1/CAMTA3 during Increased Temperature Resulting in Compromised Immune Response in Plants

Int. J. Mol. Sci. 2022, 23(4), 2175; https://doi.org/10.3390/ijms23042175
by Peiguo Yuan and B. W. Poovaiah *
Reviewer 1: Anonymous
Reviewer 2: Anonymous
Int. J. Mol. Sci. 2022, 23(4), 2175; https://doi.org/10.3390/ijms23042175
Submission received: 27 January 2022 / Accepted: 12 February 2022 / Published: 16 February 2022
(This article belongs to the Special Issue Plants Responses to Climate Change)

Round 1

Reviewer 1 Report

The present article "Interplay between Ca2+/calmodulin-mediated signaling and AtSR1/CAMTA3 during increased temperature resulting in compromised immune response in plants" designed on a good theme and  well presented. The article can be accepted for publication in this journal.

Reviewer 2 Report

It is evident that behind this manuscript a lot of work has been done and I encourage the authors to proceed with their work. The impact of increased temperature was examined on some aspects of plant defence system during pathogen entry and growth.  Stomatal defence and apoplastic immune response were shown to be influenced by elevated temperature. Information gained by this research I find very beneficial and valuable. It would be interesting to investigate in some future studies does and how the mechanisms related to AtSR1  and SA are affected in some other induced stress conditions beside elevated temperature, and how does it affect the susceptibility to some other pathogens, other then bacteria. The manuscript is well written, easy to follow, very logical from the idea to the realization and presentation, therefore I consider this work worth-publishing and propose to be accepted in present form.

Back to TopTop