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Keywords = Giovanni Andrea Gilio

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19 pages, 5966 KiB  
Article
“But Angels Don’t Have Wings”: Art, Religion, and Michelangelo’s Last Judgment in Gilio’s Dialogue on the Errors and Abuses of Painters
by Anthony Presti Russell
Religions 2023, 14(12), 1486; https://doi.org/10.3390/rel14121486 - 29 Nov 2023
Viewed by 4975
Abstract
This article provides a close reading of Giovanni Andrea Gilio’s critique of Michelangelo’s Last Judgment as presented in the Dialogue on the Errors and Abuses of Painters (1564). The dialogue has generally been taken as reflecting the emerging Counter-Reformation concerns regarding the indecorousness [...] Read more.
This article provides a close reading of Giovanni Andrea Gilio’s critique of Michelangelo’s Last Judgment as presented in the Dialogue on the Errors and Abuses of Painters (1564). The dialogue has generally been taken as reflecting the emerging Counter-Reformation concerns regarding the indecorousness of contemporary religious art, concerns that led to the censoring of the Last Judgment’s nudes in 1564 after the Council of Trent’s decree on sacred images. One frequent justification for ecclesiastical oversight over the production of religious art was that artists such as Michelangelo had prioritized their art over its religious contents and devotional aims. Though Gilio’s work has been read as confirming this view, this essay argues that the various opinions expressed during the animated exchanges in the dialogue yield a set of nuanced and often innovative interpretations of the Last Judgment that resist a reductive dichotomy between art and religion. Whether intentionally or not, the dialogue conveys that by the time of Michelangelo, and perhaps because of Michelangelo, the forms of art and the contents of religion could not be so easily distinguished from each other, largely because the artist’s subjectivity blurred the boundaries between the two. Full article
(This article belongs to the Special Issue Religions in Ritual, Spectacle, and Drama in the Medieval & Early Modern World)
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12 pages, 1537 KiB  
Article
Partial Lipodystrophy and LMNA p.R545H Variant
by Silvia Magno, Giovanni Ceccarini, Andrea Barison, Iacopo Fabiani, Alessandro Giacomina, Donatella Gilio, Caterina Pelosini, Anna Rubegni, Michele Emdin, Gian Luca Gatti, Filippo Maria Santorelli, Maria Rita Sessa and Ferruccio Santini
J. Clin. Med. 2021, 10(5), 1142; https://doi.org/10.3390/jcm10051142 - 9 Mar 2021
Cited by 4 | Viewed by 5252
Abstract
Laminopathies are disorders caused by LMNA gene mutations, which selectively affect different tissues and organ systems, and present with heterogeneous clinical and pathological traits. The molecular mechanisms behind these clinical differences and tissue specificity have not been fully clarified. We herein examine the [...] Read more.
Laminopathies are disorders caused by LMNA gene mutations, which selectively affect different tissues and organ systems, and present with heterogeneous clinical and pathological traits. The molecular mechanisms behind these clinical differences and tissue specificity have not been fully clarified. We herein examine the case of a patient carrying a heterozygous LMNA c.1634G>A (p.R545H) variant with a mild, transient myopathy, who was referred to our center for the suspicion of lipodystrophy. At physical examination, an abnormal distribution of subcutaneous fat was noticed, with fat accumulation in the anterior regions of the neck, resembling the fat distribution pattern of familial partial lipodystrophy type 2 (FPLD2). The R545H missense variant has been found at very low allelic frequency in public databases, and in silico analysis showed that this amino acid substitution is predicted to have a damaging role. Other patients carrying the heterozygous LMNA p.R545H allele have shown a marked clinical heterogeneity in terms of phenotypic body fat distribution and severity of organ system involvement. These findings indicate that the LMNA p.R545H heterozygous variant exhibits incomplete penetrance and highly variable expressivity. We hypothesized that additional genetic factors, epigenetic mechanisms, or environmental triggers might explain the variable expressivity of phenotypes among various patients. Full article
(This article belongs to the Special Issue Infrequent Lipodystrophies: Challenges in Pathogenesis, Diagnosis and Treatment)
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