Next Article in Journal / Special Issue
Investigation of the Matrix Metalloproteinase-2 Gene in Patients with Non-Syndromic Mitral Valve Prolapse
Previous Article in Journal
Vascular Calcification and Stone Disease: A New Look towards the Mechanism
Previous Article in Special Issue
Altered Hemodynamics in the Embryonic Heart Affects Outflow Valve Development
Open AccessReview

Imaging of Mitral Valve Prolapse: What Can We Learn from Imaging about the Mechanism of the Disease?

Heart Institute, Ein Kerem Campus, Hadassah Hebrew University Medical Center, POB 12000, 92110 Jerusalem, Israel
*
Author to whom correspondence should be addressed.
Academic Editors: Russell Norris and Cheryl L. Maslen
J. Cardiovasc. Dev. Dis. 2015, 2(3), 165-175; https://doi.org/10.3390/jcdd2030165
Received: 28 February 2015 / Revised: 11 June 2015 / Accepted: 1 July 2015 / Published: 10 July 2015
(This article belongs to the Special Issue Genetics and Cardiovascular Development and Disease)
Mitral valve prolapse (MVP) is the most common mitral valve disorder affecting 2%–3% of the general population. Two histological forms for the disease exist: Myxomatous degeneration and fibroelastic disease. Pathological evidence suggests the disease is not confined solely to the valve tissue, and accumulation of proteoglycans and fibrotic tissue can be seen in the adjacent myocardium of MVP patients. MVP is diagnosed by demonstrating valve tissue passing the annular line into the left atrium during systole. In this review we will discuss the advantages and limitations of various imaging modalities in their MVP diagnosis ability as well as the potential for demonstrating extra associated valvular pathologies. View Full-Text
Keywords: mitral valve prolapse; imaging; myxomatous degeneration; fibroelastic deficiency mitral valve prolapse; imaging; myxomatous degeneration; fibroelastic deficiency
Show Figures

Figure 1

MDPI and ACS Style

Durst, R.; Gilon, D. Imaging of Mitral Valve Prolapse: What Can We Learn from Imaging about the Mechanism of the Disease? J. Cardiovasc. Dev. Dis. 2015, 2, 165-175.

Show more citation formats Show less citations formats

Article Access Map

1
Only visits after 24 November 2015 are recorded.
Back to TopTop