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Correction

Correction: Xin et al. Dexras1 Induces Dysdifferentiation of Oligodendrocytes and Myelin Injury by Inhibiting the cAMP-CREB Pathway after Subarachnoid Hemorrhage. Cells 2022, 11, 2976

1
Department of Neurosurgery, The First Affiliated Hospital of Chongqing Medical University, 1 Friendship Road, Chongqing 400016, China
2
Laboratory of Skeletal Development and Regeneration, Institute of Life Sciences, Chongqing Medical University, Chongqing 400016, China
3
Department of Experimental and Clinical Neuropathology, Mossakowski Medical Research Institute Polish Academy of Sciences, 02-106 Warsaw, Poland
*
Author to whom correspondence should be addressed.
Cells 2024, 13(23), 1975; https://doi.org/10.3390/cells13231975
Submission received: 22 October 2024 / Accepted: 23 October 2024 / Published: 29 November 2024

Error in Figure

In the original publication [1], there was a mistake in Figure 8D. The authors accidentally inserted the image of SAH + LV-scramble in Figure 6D into the Figure 8D SAH + 8-Bromo-cAMP image when authors were drawing. The corrected version of Figure 8 appears below.
The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Xin, Y.; Chen, J.; Zhang, H.; Ostrowski, R.P.; Liang, Y.; Zhao, J.; Xiang, X.; Liang, F.; Fu, W.; Huang, H.; et al. Dexras1 Induces Dysdifferentiation of Oligodendrocytes and Myelin Injury by Inhibiting the cAMP-CREB Pathway after Subarachnoid Hemorrhage. Cells 2022, 11, 2976. [Google Scholar] [CrossRef] [PubMed]
Figure 8. (A) Representative Western blots of Dexras1, CREB, and pCREB expression after in vitro subarachnoid hemorrhage with the activation by different 8-Bromo-cAMP sodium salts; (B) Western blot analysis of Dexras1, CREB, and pCREB. Results are percentages relative to β-actin levels. (* p < 0.05); (C) the results of ELISA used to detect IL-1β and TNF-α levels, (** p < 0.01); (D) immunofluorescence detection of oligodendrocyte precursor cell line differentiation after in vitro subarachnoid hemorrhage in each group, co-culture of SAH+8-Bromo-cAMP group neurons and oligodendrocyte cells treated with IGF−1 (a), co-culture of SAH+8BMP+Dexras1+group neurons and oligodendrocyte cells treated with IGF−1 (b), co-culture of SAH+8-Bromo-cAMP+Dexras1group neurons and oligodendrocyte cells treated with IGF−1 (c), OPCs (NG2, red), OLGs (CNPase, green). Scale bars: ea = 25 μm. (* p < 0.05, ** p < 0.01); (E) representative Western blots of Dexras1, CREB, and pCREB after 8-Bromo-cAMP sodium salt treatment combined with Dexras1 overexpression or knockdown after subarachnoid hemorrhage in vitro; (F) Western blot semi-quantitative analysis of Dexras1, CREB, and pCRE. Results are percentages relative to β-actin levels. (* p < 0.05, ** p < 0.01, *** p < 0.001) (G). ELISA results show IL-1β and TNF-α levels. (** p < 0.01).
Figure 8. (A) Representative Western blots of Dexras1, CREB, and pCREB expression after in vitro subarachnoid hemorrhage with the activation by different 8-Bromo-cAMP sodium salts; (B) Western blot analysis of Dexras1, CREB, and pCREB. Results are percentages relative to β-actin levels. (* p < 0.05); (C) the results of ELISA used to detect IL-1β and TNF-α levels, (** p < 0.01); (D) immunofluorescence detection of oligodendrocyte precursor cell line differentiation after in vitro subarachnoid hemorrhage in each group, co-culture of SAH+8-Bromo-cAMP group neurons and oligodendrocyte cells treated with IGF−1 (a), co-culture of SAH+8BMP+Dexras1+group neurons and oligodendrocyte cells treated with IGF−1 (b), co-culture of SAH+8-Bromo-cAMP+Dexras1group neurons and oligodendrocyte cells treated with IGF−1 (c), OPCs (NG2, red), OLGs (CNPase, green). Scale bars: ea = 25 μm. (* p < 0.05, ** p < 0.01); (E) representative Western blots of Dexras1, CREB, and pCREB after 8-Bromo-cAMP sodium salt treatment combined with Dexras1 overexpression or knockdown after subarachnoid hemorrhage in vitro; (F) Western blot semi-quantitative analysis of Dexras1, CREB, and pCRE. Results are percentages relative to β-actin levels. (* p < 0.05, ** p < 0.01, *** p < 0.001) (G). ELISA results show IL-1β and TNF-α levels. (** p < 0.01).
Cells 13 01975 g008
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MDPI and ACS Style

Xin, Y.; Chen, J.; Zhang, H.; Ostrowski, R.P.; Liang, Y.; Zhao, J.; Xiang, X.; Liang, F.; Fu, W.; Huang, H.; et al. Correction: Xin et al. Dexras1 Induces Dysdifferentiation of Oligodendrocytes and Myelin Injury by Inhibiting the cAMP-CREB Pathway after Subarachnoid Hemorrhage. Cells 2022, 11, 2976. Cells 2024, 13, 1975. https://doi.org/10.3390/cells13231975

AMA Style

Xin Y, Chen J, Zhang H, Ostrowski RP, Liang Y, Zhao J, Xiang X, Liang F, Fu W, Huang H, et al. Correction: Xin et al. Dexras1 Induces Dysdifferentiation of Oligodendrocytes and Myelin Injury by Inhibiting the cAMP-CREB Pathway after Subarachnoid Hemorrhage. Cells 2022, 11, 2976. Cells. 2024; 13(23):1975. https://doi.org/10.3390/cells13231975

Chicago/Turabian Style

Xin, Yuanjun, Jie Chen, Hongxia Zhang, Robert P. Ostrowski, Yidan Liang, Jun Zhao, Xiang Xiang, Fuming Liang, Wenqiao Fu, Hao Huang, and et al. 2024. "Correction: Xin et al. Dexras1 Induces Dysdifferentiation of Oligodendrocytes and Myelin Injury by Inhibiting the cAMP-CREB Pathway after Subarachnoid Hemorrhage. Cells 2022, 11, 2976" Cells 13, no. 23: 1975. https://doi.org/10.3390/cells13231975

APA Style

Xin, Y., Chen, J., Zhang, H., Ostrowski, R. P., Liang, Y., Zhao, J., Xiang, X., Liang, F., Fu, W., Huang, H., Wu, X., Su, J., Deng, J., & He, Z. (2024). Correction: Xin et al. Dexras1 Induces Dysdifferentiation of Oligodendrocytes and Myelin Injury by Inhibiting the cAMP-CREB Pathway after Subarachnoid Hemorrhage. Cells 2022, 11, 2976. Cells, 13(23), 1975. https://doi.org/10.3390/cells13231975

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