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Influenza Virus Infection, Interferon Response, Viral Counter-Response, and Apoptosis

1
Institut Pasteur Korea, Gyeonggi-do 13488, Korea
2
Institute of Technology, University of Tartu, Tartu 50090, Estonia
3
Department of Clinical and Molecular Medicine, Norwegian University of Science and Technology, Trondheim 7028, Norway
*
Author to whom correspondence should be addressed.
Academic Editor: Marc Kvansakul
Viruses 2017, 9(8), 223; https://doi.org/10.3390/v9080223
Received: 30 June 2017 / Revised: 27 July 2017 / Accepted: 8 August 2017 / Published: 12 August 2017
(This article belongs to the Special Issue Viral Infection and Apoptosis)
Human influenza A viruses (IAVs) cause global pandemics and epidemics, which remain serious threats to public health because of the shortage of effective means of control. To combat the surge of viral outbreaks, new treatments are urgently needed. Developing new virus control modalities requires better understanding of virus-host interactions. Here, we describe how IAV infection triggers cellular apoptosis and how this process can be exploited towards the development of new therapeutics, which might be more effective than the currently available anti-influenza drugs. View Full-Text
Keywords: influenza virus; apoptosis; antiviral agent; innate immunity; host response influenza virus; apoptosis; antiviral agent; innate immunity; host response
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MDPI and ACS Style

Shim, J.M.; Kim, J.; Tenson, T.; Min, J.-Y.; Kainov, D.E. Influenza Virus Infection, Interferon Response, Viral Counter-Response, and Apoptosis. Viruses 2017, 9, 223.

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