Li, S.; Wu, C.; Zhu, L.; Gao, J.; Fang, J.; Li, D.; Fu, M.; Liang, R.; Wang, L.; Cheng, M.;
et al. By Improving Regional Cortical Blood Flow, Attenuating Mitochondrial Dysfunction and Sequential Apoptosis Galangin Acts as a Potential Neuroprotective Agent after Acute Ischemic Stroke. Molecules 2012, 17, 13403-13423.
https://doi.org/10.3390/molecules171113403
AMA Style
Li S, Wu C, Zhu L, Gao J, Fang J, Li D, Fu M, Liang R, Wang L, Cheng M,
et al. By Improving Regional Cortical Blood Flow, Attenuating Mitochondrial Dysfunction and Sequential Apoptosis Galangin Acts as a Potential Neuroprotective Agent after Acute Ischemic Stroke. Molecules. 2012; 17(11):13403-13423.
https://doi.org/10.3390/molecules171113403
Chicago/Turabian Style
Li, Shaojing, Chuanhong Wu, Li Zhu, Jian Gao, Jing Fang, Defeng Li, Meihong Fu, Rixin Liang, Lan Wang, Ming Cheng,
and et al. 2012. "By Improving Regional Cortical Blood Flow, Attenuating Mitochondrial Dysfunction and Sequential Apoptosis Galangin Acts as a Potential Neuroprotective Agent after Acute Ischemic Stroke" Molecules 17, no. 11: 13403-13423.
https://doi.org/10.3390/molecules171113403
APA Style
Li, S., Wu, C., Zhu, L., Gao, J., Fang, J., Li, D., Fu, M., Liang, R., Wang, L., Cheng, M., & Yang, H.
(2012). By Improving Regional Cortical Blood Flow, Attenuating Mitochondrial Dysfunction and Sequential Apoptosis Galangin Acts as a Potential Neuroprotective Agent after Acute Ischemic Stroke. Molecules, 17(11), 13403-13423.
https://doi.org/10.3390/molecules171113403
