Ono, N.; Fujita, T.; Miki, M.; Nishiyama, K.; Izawa, T.; Aoyama, T.; Kuwamura, M.; Fujii, H.; Azuma, Y.-T.
Interleukin-19 Gene-Deficient Mice Promote Liver Fibrosis via Enhanced TGF-β Signaling, and the Interleukin-19-CCL2 Axis Is Important in the Direction of Liver Fibrosis. Biomedicines 2023, 11, 2064.
https://doi.org/10.3390/biomedicines11072064
AMA Style
Ono N, Fujita T, Miki M, Nishiyama K, Izawa T, Aoyama T, Kuwamura M, Fujii H, Azuma Y-T.
Interleukin-19 Gene-Deficient Mice Promote Liver Fibrosis via Enhanced TGF-β Signaling, and the Interleukin-19-CCL2 Axis Is Important in the Direction of Liver Fibrosis. Biomedicines. 2023; 11(7):2064.
https://doi.org/10.3390/biomedicines11072064
Chicago/Turabian Style
Ono, Naoshige, Takashi Fujita, Mariko Miki, Kazuhiro Nishiyama, Takeshi Izawa, Tomoko Aoyama, Mitsuru Kuwamura, Hideki Fujii, and Yasu-Taka Azuma.
2023. "Interleukin-19 Gene-Deficient Mice Promote Liver Fibrosis via Enhanced TGF-β Signaling, and the Interleukin-19-CCL2 Axis Is Important in the Direction of Liver Fibrosis" Biomedicines 11, no. 7: 2064.
https://doi.org/10.3390/biomedicines11072064
APA Style
Ono, N., Fujita, T., Miki, M., Nishiyama, K., Izawa, T., Aoyama, T., Kuwamura, M., Fujii, H., & Azuma, Y.-T.
(2023). Interleukin-19 Gene-Deficient Mice Promote Liver Fibrosis via Enhanced TGF-β Signaling, and the Interleukin-19-CCL2 Axis Is Important in the Direction of Liver Fibrosis. Biomedicines, 11(7), 2064.
https://doi.org/10.3390/biomedicines11072064
