Redox Balance in Kidney Disease
A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "Health Outcomes of Antioxidants and Oxidative Stress".
Deadline for manuscript submissions: closed (15 August 2023) | Viewed by 6626
Special Issue Editor
Interests: hemodialysis; dialysis; chronic renal failure; hypertension; cardiovascular disease; diabetes; clinical nephrology; acute kidney injury; chronic kidney failure; kidney transplantation
Special Issue Information
Dear Colleagues,
Oxidative stress can damage cells, proteins, and DNA, which can contribute to aging, a process characterized by the progressive loss of tissue and organ function. Oxidative stress is the result of an imbalance between reactive oxygen and nitrogen species (RONS) production and antioxidant defenses.
Oxidative stress has been found in kidney disease due to both antioxidant deficiency and increased RONS production. The kidney is a high-metabolic-rate organ where many oxidative reactions occur, making it susceptible to damage from RONS.
Several studies have shown that RONS can accelerate the progression of kidney disease. In patients with advanced stages of chronic kidney disease (CKD), increased RONS levels are associated with complications such as inflammation, anemia, premature vascular aging, hypertension, and atherosclerosis.
This Special Issue will discuss recent findings on oxidative stress biomarkers and pathophysiological processes, as well as how oxidative stress affects renal function and promotes damage in each nephron segment, including renal vessels, glomeruli and tubules.
We invite you to submit your latest research or a review article for this Special Issue, summarizing current research on oxidative stress in acute and chronic kidney disease.
Prof. Dr. Robert Ekart
Guest Editor
Manuscript Submission Information
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Keywords
- oxidative stress
- acute and chronic kidney disease
- reactive oxygen and nitrogen species
- uremic toxins
- premature aging