Oxidative Stress in Striated Muscle and Other Tissues
A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "Health Outcomes of Antioxidants and Oxidative Stress".
Deadline for manuscript submissions: 30 November 2024 | Viewed by 40
Special Issue Editor
Interests: skeletal muscle physiology; excitation–transcription coupling; gene expression; muscle metabolism
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
ROS play an important signaling role in skeletal and cardiac muscles, as well as in other tissues closely associated with calcium signals that may be or may be not involved in muscle contraction. Several signaling pathways in striated muscle can be activated by an increase in reactive oxygen species (ROS) and reactive nitrogen species (RNS) production. The large magnitude of calcium signals involved in both the contractile process and the deleterious processes induced by excess ROS/RNS production has made the study of the physiological role of ROS difficult and has restricted our in-depth research of these events for many years. Abnormal ROS/RNS production appears to be involved in several striated muscle-related diseases, including muscle wasting, muscular dystrophies, aging-related sarcopenia, cardiac wasting and cancer cachexia. Metabolic diseases such as obesity are also related to abnormal ROS/RNS handling by muscle cells, leading to insulin resistance and T2D.
We invite you to submit your latest research findings or a review article to this Special Issue, which will collate current research in both striated muscle and exercise concerning ROS/RNS production, ROS/RNS regulation and ROS-/RNS-related deleterious processes and diseases. Collating this new knowledge of ROS/RNS homeostasis in striated muscle will provide important insights into the fine-tuning and physiological impact of important signaling within muscle cells.
Prof. Dr. Enrique Jaimovich
Guest Editor
Manuscript Submission Information
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Keywords
- ROS/RNS production
- mitochondria ROS
- NADPH oxidase
- metabolic regulation
- cardiac oxidative stress
- ROS sources
- exercise-induced ROS/RNS
- antioxidants
- ROS/RNS in disease
