State-of-Art in Autophagy and Neurodegeneration

Dear Colleagues, 

Autophagy suppression was shown in several neurodegenerative diseases, like Alzheimer's, Parkinson's, and Huntington's diseases connected with the accumulation of amyloid-β, α-synuclein, and huntingtin, respectively.

Autophagy is also reduced in aging, which associates with physiological dementia, neuronal integrity loss and increased susceptibility to neurodegenerative disorders including Alzheimer's Disease, Parkinson Disease and Frontotemporal dementia, to name a few.

Thus, the autophagy activation through different pathways can reverse both neuronal dopaminergic and behavioral deficits in vivo and seems to be a promising therapy in pathology. Induction of autophagy as a means of improving the viability of cells and organisms is an attractive approach to experimental therapy of neurodegeneration.

This Special Issue of Cells will present research articles and reviews that cover the scope of autophagy and neurodegeneration. All scientists working in these fields are cordially invited to submit their manuscripts.

Prof. Dr. Ciro Isidoro
Prof. Dr. Tatyana A. Korolenko
Guest Editors

Manuscript Submission Information

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Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-anonymized peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Cells is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.