The “Dead-End Tract” and Its Role in Arrhythmogenesis
AbstractIdiopathic outflow tract ventricular arrhythmias (VAs) represent a significant proportion of all VAs. The mechanism is thought to be catecholamine-mediated delayed after depolarizations and triggered activity, although other etiologies should be considered. In the adult cardiac conduction system it has been demonstrated that sometimes an embryonic branch, the so-called “dead-end tract”, persists beyond the bifurcation of the right and left bundle branch (LBB). Several findings suggest an involvement of this tract in idiopathic VAs (IVAs). The aim of this review is to summarize our current knowledge and the possible clinical significance of this tract. View Full-Text
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de Vries, L.; Hendriks, A.; Szili-Torok, T. The “Dead-End Tract” and Its Role in Arrhythmogenesis. J. Cardiovasc. Dev. Dis. 2016, 3, 11.
de Vries L, Hendriks A, Szili-Torok T. The “Dead-End Tract” and Its Role in Arrhythmogenesis. Journal of Cardiovascular Development and Disease. 2016; 3(2):11.Chicago/Turabian Style
de Vries, Lennart; Hendriks, Astrid; Szili-Torok, Tamas. 2016. "The “Dead-End Tract” and Its Role in Arrhythmogenesis." J. Cardiovasc. Dev. Dis. 3, no. 2: 11.
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