Keywordsdiphtheria; diphtheria toxin; cellular uptake; membrane transport; EGA; C2-toxin; iota-toxin; binding components; chloroquine; black lipid bilayer; aminoquinolinium salts; multivalency; planar lipid bilayer technique; Bacillus anthracis; protective antigen; pore blockage; Clostridium perfringens; intestinal infections; gas gangrene; toxins; sialidases; sialidase inhibitors; botulinum toxin; foodborne toxins; probiotic bacteria; toxin absorption; endocytosis; Ras; C. difficile Toxin B; mono-O-glucosylation; p21-activated kinase; actin; Shiga toxin; Stx1; Stx2; hemolytic uremic syndrome; inhibitors; chloroquine; fluorodeoxyglucose; Mn2+; n/a